Schmid P
Notfallzentrum, Chirurgie, Inselspital Bern.
Schweiz Med Wochenschr. 1999 Sep 25;129(38):1368-80.
Whiplash-associated disorders (WAD) represent a class of clinical complaints which commonly result from rear-end car accidents. An automobile collision can generate major forces which are transferred to the neck by an acceleration-deceleration mechanism (whiplash), resulting in bony or soft-tissue injuries (whiplash injury). Incidence of WAD is estimated to be 0.1 to 3.8/1000/year; WAD cost $29 billion a year in the USA. They can be classified clinically into 5 degrees of severity, namely WAD grades 0 to IV. Signs and symptoms typically crescendo during the first few days after an accident. Pathological findings (especially of musculo-skeletal or neurological types) must often be sought actively and should be documented at the earliest stage. Prevention of possible chronicity is the most important goal in clinical management of WAD. WAD grade IV patients are treated in the way their fracture or dislocation demands. Therapy of WAD grades I to III has three main aspects: non-narcotic analgesics, early active mobilisation (to the extent possible consistent with pain) and education of the patient. Soft collars should not be used (or only temporarily and sparingly). Most patients with WAD grades I-III feel well again relatively soon. Symptoms and signs that persist for longer than two months are important warning signs for imminent chronicity, which occurs at rates of 14-42%. In such cases, an interdisciplinary approach is recommended. Risk factors are accident severity, head position at the time of accident, age and pretraumatic existence of headache. Patients with chronic complaints can develop additional psychic and cognitive problems, which are caused by--and not the cause of--their chronic disorder. Therapy of chronic whiplash-associated disorders involves all the problems inherent in therapies of chronic pain. There are many therapeutic concepts, but little evidence that anything helps. Prevention of whiplash injuries is therefore very important in view of the lack of powerful treatment options. Although there is a substantial body of scientific literature about WAD, many unanswered questions remain. In particular the most important questions (how can patients with acute and chronic disorders be helped best) have no clear answer yet. Furthermore, there are many opinions and prejudices (especially concerning psycho-social factors of WAD) which have no scientific basis. Therefore, an intensive exchange of information between health care professionals, patients and the general public appears to be very important.
挥鞭样相关疾病(WAD)是一类常见于汽车追尾事故后的临床症状。汽车碰撞会产生巨大力量,通过加速-减速机制(挥鞭样动作)传递至颈部,导致骨骼或软组织损伤(挥鞭样损伤)。据估计,WAD的发病率为每年0.1至3.8/1000;在美国,WAD每年造成的损失达290亿美元。临床上可将其分为5个严重程度等级,即WAD 0至IV级。事故后的头几天,症状和体征通常会逐渐加重。必须经常积极寻找病理结果(尤其是肌肉骨骼或神经类型的),并应在最早阶段记录下来。预防可能出现的慢性化是WAD临床管理中最重要的目标。IV级WAD患者按其骨折或脱位的治疗要求进行治疗。I至III级WAD的治疗有三个主要方面:非麻醉性镇痛药、早期积极活动(在疼痛允许的范围内)以及对患者进行教育。不应使用软颈托(或仅临时少量使用)。大多数I至III级WAD患者相对较快就能恢复良好。症状和体征持续超过两个月是即将出现慢性化的重要警示信号,慢性化发生率为14%至42%。在这种情况下,建议采用多学科方法。危险因素包括事故严重程度、事故发生时头部位置、年龄以及创伤前是否存在头痛。患有慢性症状的患者可能会出现额外的心理和认知问题,这些问题是由其慢性疾病引起的,而非慢性疾病的病因。慢性挥鞭样相关疾病的治疗涉及慢性疼痛治疗中固有的所有问题。有许多治疗理念,但几乎没有证据表明有什么方法有效。鉴于缺乏有效的治疗选择,预防挥鞭样损伤因此非常重要。尽管有大量关于WAD的科学文献,但仍有许多问题未得到解答。特别是最重要的问题(如何最好地帮助急性和慢性疾病患者)尚无明确答案。此外,存在许多没有科学依据的观点和偏见(尤其是关于WAD的心理社会因素)。因此,医疗保健专业人员、患者和公众之间进行深入的信息交流显得非常重要。