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A role for nitric oxide in hydroxyurea-mediated fetal hemoglobin induction.

作者信息

King S Bruce

机构信息

Department of Chemistry, Wake Forest University, Winston-Salem, North Carolina 27109, USA.

出版信息

J Clin Invest. 2003 Jan;111(2):171-2. doi: 10.1172/JCI17597.

Abstract
摘要

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本文引用的文献

1
HYDROXYUREA: MECHANISM OF ACTION.
Science. 1963 Nov 22;142(3595):1069-70. doi: 10.1126/science.142.3595.1069.
3
Cell-free hemoglobin limits nitric oxide bioavailability in sickle-cell disease.
Nat Med. 2002 Dec;8(12):1383-9. doi: 10.1038/nm1202-799. Epub 2002 Nov 11.
4
Horseradish peroxidase catalyzed nitric oxide formation from hydroxyurea.
J Am Chem Soc. 2002 Apr 3;124(13):3473-80. doi: 10.1021/ja012271v.
5
Nitric oxide donor properties of hydroxyurea in patients with sickle cell disease.
Br J Haematol. 2002 Feb;116(2):436-44. doi: 10.1046/j.1365-2141.2002.03274.x.
6
Iron nitrosyl hemoglobin formation from the reactions of hemoglobin and hydroxyurea.
Biochemistry. 2002 Feb 19;41(7):2466-74. doi: 10.1021/bi011470o.
7
Nitric oxide therapy in sickle cell disease.
Semin Hematol. 2001 Oct;38(4):333-42. doi: 10.1016/s0037-1963(01)90027-7.
9
Mechanism for fetal globin gene expression: role of the soluble guanylate cyclase-cGMP-dependent protein kinase pathway.
Proc Natl Acad Sci U S A. 2001 Feb 13;98(4):1847-52. doi: 10.1073/pnas.98.4.1847. Epub 2001 Feb 6.

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