Engelhardt P
Med Klin. 1976 Apr 23;71(17):706-10.
Etiology of idiopathic facial paralysis remains mostly unknown because further investigations seem unnecessary being the only symptom. Differentiated evaluation of CSF however for cytological or proteinous abnormalities should be performed aside serological examinations. In 9 patients treated within 8 months in our hospital diagnosis could be made by these procedures. Inflammation, if cause of facial paralysis, can only call pleocytosis, if localised within or next to the leptomeninges; protein of CSF perhaps will increase, if local inflammation of the nerve is more distant to subarachnoid space. "Idiopathic facial paralysis" however will not exclude focal inflammation far from subarachnoid space. Surgical decompression should not be performed without previous examination of CSF in regard of it's uncertain success.
特发性面神经麻痹的病因大多仍不清楚,因为作为唯一症状似乎无需进一步调查。然而,除血清学检查外,还应对脑脊液进行细胞学或蛋白质异常的鉴别评估。在我院8个月内治疗的9例患者中,通过这些检查得以确诊。如果炎症是面神经麻痹的病因,只有当炎症局限于软脑膜内或其附近时才会引起脑脊液细胞数增多;如果神经的局部炎症距离蛛网膜下腔较远,脑脊液蛋白可能会增加。然而,“特发性面神经麻痹”并不排除远离蛛网膜下腔的局灶性炎症。鉴于手术减压成功率不确定,在未事先检查脑脊液的情况下不应进行。