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Glutamatergic synaptic dysfunction in hyperammonemic syndromes.

作者信息

Rao V L, Murthy C R, Butterworth R F

机构信息

School of Life Sciences, University of Hyderabad, India.

出版信息

Metab Brain Dis. 1992 Mar;7(1):1-20. doi: 10.1007/BF01000437.

DOI:10.1007/BF01000437

PMID:1608363

Abstract

摘要

相似文献

Glutamatergic synaptic dysfunction in hyperammonemic syndromes.

Metab Brain Dis. 1992 Mar;7(1):1-20. doi: 10.1007/BF01000437.

7 ISN special neurochemistry conference 'Synaptic function and dysfunction in brain diseases'.

J Neurochem. 2016 Dec;139(6):918-920. doi: 10.1111/jnc.13839. Epub 2016 Nov 16.

Urea cycle disorders: clinical paradigm of hyperammonemic encephalopathy.

Prog Liver Dis. 1995;13:293-309.

Current status of hyperammonemic syndromes.

Hepatology. 1982 Jul-Aug;2(4):495-506. doi: 10.1002/hep.1840020417.

Block of glutamatergic excitatory synaptic channels by chlorisondamine.

Mol Pharmacol. 1981 Mar;19(2):349-53.

The glutamatergic nerve terminal.

Eur J Biochem. 1993 Mar 15;212(3):613-31. doi: 10.1111/j.1432-1033.1993.tb17700.x.

[Ammonia content of the brain, liver and blood of rats in radiation injury].

Radiobiologiia. 1987 Jan-Feb;27(1):112-5.

Glutamatergic synapses and glutamate receptors on locust muscle.

J Physiol (Paris). 1979;75(6):615-21.

Functional diversity of central glutamate synapses--pre- and post-synaptic mechanisms.

Acta Physiol Scand. 1994 Jan;150(1):1-10. doi: 10.1111/j.1748-1716.1994.tb09653.x.

[Role of dopamine receptors in regulating glutamatergic synaptic transmission in the rat neostriatum].

Fiziol Zh SSSR Im I M Sechenova. 1981 Mar;67(3):384-9.

引用本文的文献

Taurine supplementation abates cirrhosis-associated locomotor dysfunction.

Clin Exp Hepatol. 2018 Jun;4(2):72-82. doi: 10.5114/ceh.2018.75956. Epub 2018 May 25.

Hyperammonia induces specific liver injury through an intrinsic Ca2+-independent apoptosis pathway.

BMC Gastroenterol. 2014 Aug 22;14:151. doi: 10.1186/1471-230X-14-151.

Brain edema in acute liver failure: inhibition by L-histidine.

Am J Pathol. 2010 Mar;176(3):1400-8. doi: 10.2353/ajpath.2010.090756. Epub 2010 Jan 14.

Signaling factors in the mechanism of ammonia neurotoxicity.

Metab Brain Dis. 2009 Mar;24(1):103-17. doi: 10.1007/s11011-008-9113-6. Epub 2008 Dec 23.

Altered modulation of motor activity by group I metabotropic glutamate receptors in the nucleus accumbens in hyperammonemic rats.

Metab Brain Dis. 2005 Dec;20(4):347-58. doi: 10.1007/s11011-005-7918-0.

Contribution of extracellular glutamine as an anaplerotic substrate to neuronal metabolism: a re-evaluation by multinuclear NMR spectroscopy in primary cultured neurons.

Neurochem Res. 2005 Oct;30(10):1269-81. doi: 10.1007/s11064-005-8798-8.

Altered modulation of soluble guanylate cyclase by nitric oxide in patients with liver disease.

Metab Brain Dis. 2002 Dec;17(4):295-301. doi: 10.1023/a:1021953717331.

Neuronal nitric oxide synthase and Hepatic Encephalopathy.

Metab Brain Dis. 1998 Sep;13(3):175-89. doi: 10.1023/a:1023281307660.

Plasma concentrations of endogenous benzodiazepine-receptor ligands in patients with hepatic encephalopathy: a comparative study.

J Psychiatry Neurosci. 1998 Sep;23(4):217-22.

Effects of hyperammonaemia on brain function.

J Inherit Metab Dis. 1998;21 Suppl 1:6-20. doi: 10.1023/a:1005393104494.

本文引用的文献

Elevation of transamination of branched chain amino acids in brain in acute ammonia toxicity.

Neurochem Int. 1985;7(6):1027-31. doi: 10.1016/0197-0186(85)90151-2.

Acute metabolic effects of ammonia on the enzymes of glutamate metabolism in isolated astroglial cells.

Neurochem Int. 1983;5(5):593-7. doi: 10.1016/0197-0186(83)90051-7.

Metabolic compartments in vivo. Ammonia and glutamic acid metabolism in brain and liver.

J Biol Chem. 1962 Aug;237:2562-9.

Studies on blood ammonia in normal and shock states.

Surgery. 1953 Jul;34(1):1-8.

Kinetics of cerebral uptake processes in vitro of L-glutamine, branched-chain L-amino acids, and L-phenylalanine: effects of ouabain.

J Neurochem. 1980 Jul;35(1):67-77. doi: 10.1111/j.1471-4159.1980.tb12490.x.

Psychomotor performance defects in cirrhotic patients without overt encephalopathy.

Arch Intern Med. 1980 Apr;140(4):519-21.

Effect of acute ammonia intoxication on energy stores in the cerebral reticular activating system.

Exp Brain Res. 1981;44(3):325-30. doi: 10.1007/BF00236570.

Control of glutaminase activity in rat brain cortex in vitro: influence of glutamate, phosphate, ammonium, calcium and hydrogen ions.

Brain Res. 1981 Mar 16;208(2):363-77. doi: 10.1016/0006-8993(81)90564-3.

Current status of hyperammonemic syndromes.

Hepatology. 1982 Jul-Aug;2(4):495-506. doi: 10.1002/hep.1840020417.

Characterization of uptake and release processes for D- and L-aspartate in primary cultures of astrocytes and cerebellar granule cells.

Neurochem Res. 1983 Feb;8(2):231-43. doi: 10.1007/BF00963923.

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Glutamatergic synaptic dysfunction in hyperammonemic syndromes.

作者信息

Rao V L, Murthy C R, Butterworth R F

机构信息

School of Life Sciences, University of Hyderabad, India.

出版信息

Metab Brain Dis. 1992 Mar;7(1):1-20. doi: 10.1007/BF01000437.

DOI:10.1007/BF01000437

PMID:1608363

Abstract

摘要

相似文献

Glutamatergic synaptic dysfunction in hyperammonemic syndromes.

Metab Brain Dis. 1992 Mar;7(1):1-20. doi: 10.1007/BF01000437.

7 ISN special neurochemistry conference 'Synaptic function and dysfunction in brain diseases'.

J Neurochem. 2016 Dec;139(6):918-920. doi: 10.1111/jnc.13839. Epub 2016 Nov 16.

Urea cycle disorders: clinical paradigm of hyperammonemic encephalopathy.

Prog Liver Dis. 1995;13:293-309.

Current status of hyperammonemic syndromes.

Hepatology. 1982 Jul-Aug;2(4):495-506. doi: 10.1002/hep.1840020417.

Block of glutamatergic excitatory synaptic channels by chlorisondamine.

Mol Pharmacol. 1981 Mar;19(2):349-53.

The glutamatergic nerve terminal.

Eur J Biochem. 1993 Mar 15;212(3):613-31. doi: 10.1111/j.1432-1033.1993.tb17700.x.

[Ammonia content of the brain, liver and blood of rats in radiation injury].

Radiobiologiia. 1987 Jan-Feb;27(1):112-5.

Glutamatergic synapses and glutamate receptors on locust muscle.

J Physiol (Paris). 1979;75(6):615-21.

Functional diversity of central glutamate synapses--pre- and post-synaptic mechanisms.

Acta Physiol Scand. 1994 Jan;150(1):1-10. doi: 10.1111/j.1748-1716.1994.tb09653.x.

[Role of dopamine receptors in regulating glutamatergic synaptic transmission in the rat neostriatum].

Fiziol Zh SSSR Im I M Sechenova. 1981 Mar;67(3):384-9.

引用本文的文献

Taurine supplementation abates cirrhosis-associated locomotor dysfunction.

Clin Exp Hepatol. 2018 Jun;4(2):72-82. doi: 10.5114/ceh.2018.75956. Epub 2018 May 25.

Hyperammonia induces specific liver injury through an intrinsic Ca2+-independent apoptosis pathway.

BMC Gastroenterol. 2014 Aug 22;14:151. doi: 10.1186/1471-230X-14-151.

Brain edema in acute liver failure: inhibition by L-histidine.

Am J Pathol. 2010 Mar;176(3):1400-8. doi: 10.2353/ajpath.2010.090756. Epub 2010 Jan 14.

Signaling factors in the mechanism of ammonia neurotoxicity.

Metab Brain Dis. 2009 Mar;24(1):103-17. doi: 10.1007/s11011-008-9113-6. Epub 2008 Dec 23.

Altered modulation of motor activity by group I metabotropic glutamate receptors in the nucleus accumbens in hyperammonemic rats.

Metab Brain Dis. 2005 Dec;20(4):347-58. doi: 10.1007/s11011-005-7918-0.

Contribution of extracellular glutamine as an anaplerotic substrate to neuronal metabolism: a re-evaluation by multinuclear NMR spectroscopy in primary cultured neurons.

Neurochem Res. 2005 Oct;30(10):1269-81. doi: 10.1007/s11064-005-8798-8.

Altered modulation of soluble guanylate cyclase by nitric oxide in patients with liver disease.

Metab Brain Dis. 2002 Dec;17(4):295-301. doi: 10.1023/a:1021953717331.

Neuronal nitric oxide synthase and Hepatic Encephalopathy.

Metab Brain Dis. 1998 Sep;13(3):175-89. doi: 10.1023/a:1023281307660.

Plasma concentrations of endogenous benzodiazepine-receptor ligands in patients with hepatic encephalopathy: a comparative study.

J Psychiatry Neurosci. 1998 Sep;23(4):217-22.

Effects of hyperammonaemia on brain function.

J Inherit Metab Dis. 1998;21 Suppl 1:6-20. doi: 10.1023/a:1005393104494.

本文引用的文献

Elevation of transamination of branched chain amino acids in brain in acute ammonia toxicity.

Neurochem Int. 1985;7(6):1027-31. doi: 10.1016/0197-0186(85)90151-2.

Acute metabolic effects of ammonia on the enzymes of glutamate metabolism in isolated astroglial cells.

Neurochem Int. 1983;5(5):593-7. doi: 10.1016/0197-0186(83)90051-7.

Metabolic compartments in vivo. Ammonia and glutamic acid metabolism in brain and liver.

J Biol Chem. 1962 Aug;237:2562-9.

Studies on blood ammonia in normal and shock states.

Surgery. 1953 Jul;34(1):1-8.

Kinetics of cerebral uptake processes in vitro of L-glutamine, branched-chain L-amino acids, and L-phenylalanine: effects of ouabain.

J Neurochem. 1980 Jul;35(1):67-77. doi: 10.1111/j.1471-4159.1980.tb12490.x.

Psychomotor performance defects in cirrhotic patients without overt encephalopathy.

Arch Intern Med. 1980 Apr;140(4):519-21.

Effect of acute ammonia intoxication on energy stores in the cerebral reticular activating system.

Exp Brain Res. 1981;44(3):325-30. doi: 10.1007/BF00236570.

Control of glutaminase activity in rat brain cortex in vitro: influence of glutamate, phosphate, ammonium, calcium and hydrogen ions.

Brain Res. 1981 Mar 16;208(2):363-77. doi: 10.1016/0006-8993(81)90564-3.

Current status of hyperammonemic syndromes.

Hepatology. 1982 Jul-Aug;2(4):495-506. doi: 10.1002/hep.1840020417.

Characterization of uptake and release processes for D- and L-aspartate in primary cultures of astrocytes and cerebellar granule cells.

Neurochem Res. 1983 Feb;8(2):231-43. doi: 10.1007/BF00963923.