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Function of NKG2D in natural killer cell-mediated rejection of mouse bone marrow grafts.
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The requirement for NKG2D in NK cell-mediated rejection of parental bone marrow grafts is determined by MHC class I expressed by the graft recipient.
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NK cells rapidly reject allogeneic bone marrow in the spleen through a perforin- and Ly49D-dependent, but NKG2D-independent mechanism.
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[Effects of NKG2D and its ligands RAE-1 and H60 on graft-versus-tumor response].
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IL-21 enhances tumor rejection through a NKG2D-dependent mechanism.
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A novel ligand for the NKG2D receptor activates NK cells and macrophages and induces tumor immunity.
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Rae1 and H60 ligands of the NKG2D receptor stimulate tumour immunity.
Nature. 2001 Sep 13;413(6852):165-71. doi: 10.1038/35093109.
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Expression of NKG2D and its ligand in mouse heart allografts may have a role in acute rejection.
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Influence of SHIP on the NK repertoire and allogeneic bone marrow transplantation.
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Natural killer cells in cancer immunotherapy.
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Donor NKG2D rs1049174 polymorphism predicts hematopoietic recovery and event-free survival after single-unit cord blood transplantation in adults.
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The Role of NKG2D and Its Ligands in Autoimmune Diseases: New Targets for Immunotherapy.
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Modified dendritic cell-derived exosomes activate both NK cells and T cells through the NKG2D/NKG2D-L pathway to kill CML cells with or without T315I mutation.
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NKG2D discriminates diverse ligands through selectively mechano-regulated ligand conformational changes.
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NKG2D Natural Killer Cell Receptor-A Short Description and Potential Clinical Applications.
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The natural killer cell activating receptor, NKG2D, is critical to antibody-dependent chronic rejection in heart transplantation.
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Clonal expansion of innate and adaptive lymphocytes.
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Regulation of murine NK cell exhaustion through the activation of the DNA damage repair pathway.
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NK cell recognition of hematopoietic cells by SLAM-SAP families.
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NK cell recognition.
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Engagement of NKG2D by cognate ligand or antibody alone is insufficient to mediate costimulation of human and mouse CD8+ T cells.
J Immunol. 2005 Feb 15;174(4):1922-31. doi: 10.4049/jimmunol.174.4.1922.
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The cytomegalovirus m155 gene product subverts natural killer cell antiviral protection by disruption of H60-NKG2D interactions.
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NKG2D blockade prevents autoimmune diabetes in NOD mice.
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Roles of the NKG2D immunoreceptor and its ligands.
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INDUCTION OF IMMUNITY AND OF UNRESPONSIVENESS TO PARENTAL MARROW GRAFTS IN ADULT F-1 HYBRID MICE.
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NKG2D-mediated natural killer cell protection against cytomegalovirus is impaired by viral gp40 modulation of retinoic acid early inducible 1 gene molecules.
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NKG2D ligands: unconventional MHC class I-like molecules exploited by viruses and cancer.
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Impairment of NK cell function by NKG2D modulation in NOD mice.
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Structure and function of natural killer cell surface receptors.
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