J Athl Train. 1997 Oct;32(4):359-62.
To present the case of an 18-year-old collegiate wrestler diagnosed with vasodepressor syncope.
Vasodepressor syncope and the pathophysiologic mechanisms responsible are not fully understood. It is postulated that a sudden, rapid reduction in venous return to the heart allows for quite forceful ventricular contractions. Hypotension, bradycardia, and possible cerebral hypoxia may result, which could cause loss of consciousness.
Cardiovascular, neurologic, metabolic, and psychogenic causes.
Pharmacologic therapy of fludrocortisone acetate and potassium chloride was effective. The athlete returned to full activity without restrictions and remained symptom free throughout the remainder of the season.
Vasodepressor syncope is usually associated with full syncope. However, this 18-year-old wrestler never fully lost consciousness. His symptoms occurred primarily with exercise.
The athletic trainer should consider exercise- induced vasodepressor syncope as a possible etiology for those athletes who present with presyncopal episodes.
报告一例 18 岁大学生摔跤运动员被诊断为血管减压性晕厥的病例。
血管减压性晕厥及其相关的病理生理机制尚未完全阐明。据推测,静脉回流突然、快速减少会导致心室强烈收缩。低血压、心动过缓以及可能的脑缺氧可能会导致意识丧失。
心血管、神经、代谢和精神源性原因。
氟氢可的松醋酸盐和氯化钾的药物治疗有效。运动员恢复了全面的活动,没有限制,并且在整个赛季中都没有出现症状。
血管减压性晕厥通常与完全晕厥相关。然而,这位 18 岁的摔跤运动员从未完全失去意识。他的症状主要发生在运动时。
运动训练员应考虑运动引起的血管减压性晕厥,作为那些出现晕厥前症状的运动员的可能病因。