Reising Scott, Safford Robert, Castello Ramon, Bosworth Veronica, Freeman William, Kusumoto Fred
Department of Community Internal Medicine, Mayo Clinic, Jacksonville, Florida 32224, USA.
J Am Soc Echocardiogr. 2007 Nov;20(11):1316.e1-3. doi: 10.1016/j.echo.2007.03.003. Epub 2007 Jun 27.
Lead wire malposition is thought to be a rare complication of both permanent and temporary pacemaker implantation. The actual incidence and prevalence are unknown because of lack of reporting, which complicates consistency in treatment. Potential safeguards to prevent complications as a result of lead malposition are readily available, effective, and inexpensive, but underused. An 80-year-old white man presented to our institution with right-arm paresthesias and weakness, as well as facial numbness, 4 months after undergoing single-chamber pacemaker placement by an outside hospital because of tachybrady syndrome. Computed tomography scan of the head revealed a recent ischemic stroke. Electrocardiography revealed right bundle-branch block morphology of paced beats. Chest radiography raised the suspicion of lead malposition because of the posterior deflection of the lead wire on the lateral view. Transesophageal echocardiography conclusively demonstrated a pacemaker lead wire that transversed the aortic valve into the left ventricle without the presence of thrombus. The patient underwent successful removal of the device with a transcatheter approach, and a replacement pacemaker was placed for symptomatic bradycardia. It was recently suggested that echocardiography is not able to adequately detect thrombi on lead wires and that all patients with stroke should undergo open heart surgery for device extraction. We think that this does not adequately incorporate the significant comorbidities for some patients in the risk-benefit decision-making processes and that a transcutaneous approach is reasonable for patients without evidence of thrombi who are poor surgical candidates for an open heart procedure. A 12-lead electrocardiogram should be performed on every patient after pacemaker insertion. If right bundle-branch block morphology of paced beats is noted, chest radiography including a lateral view should be ordered. If there is any ambiguity regarding lead placement, echocardiography should be performed for determining lead malposition.
导线位置异常被认为是永久性和临时性起搏器植入术的一种罕见并发症。由于缺乏报告,实际发病率和患病率尚不清楚,这使得治疗的一致性变得复杂。预防导线位置异常导致并发症的潜在保障措施容易获得、有效且成本低廉,但未得到充分利用。一名80岁白人男性因心动过速-心动过缓综合征在外部医院接受单腔起搏器植入4个月后,出现右臂感觉异常和无力以及面部麻木,前来我院就诊。头部计算机断层扫描显示近期有缺血性中风。心电图显示起搏心律呈右束支传导阻滞形态。胸部X线检查因侧位片上导线向后偏移而怀疑导线位置异常。经食管超声心动图最终证实一根起搏器导线穿过主动脉瓣进入左心室且无血栓形成。患者通过经导管方法成功取出该装置,并因症状性心动过缓植入了替代起搏器。最近有人提出,超声心动图无法充分检测导线血栓,所有中风患者都应接受心脏直视手术取出装置。我们认为,这在风险-收益决策过程中没有充分考虑到一些患者的显著合并症,对于没有血栓证据且不适合心脏直视手术的患者,经皮方法是合理的。起搏器植入后应对每位患者进行12导联心电图检查。如果发现起搏心律呈右束支传导阻滞形态,应进行包括侧位片在内的胸部X线检查。如果导线位置存在任何疑问,应进行超声心动图检查以确定导线位置异常。