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白血病相关蛋白质降解的分子机制。

Molecular mechanisms of leukemia-associated protein degradation.

作者信息

Wu Ying-Li, Zhou Hu-Chen, Chen Guo-Qiang

机构信息

Department of Pathophysiology and Chemical Biology Division of Shanghai Universities E-Institutes, Key laboratory of Cell Differentiation and Apoptosis of the Ministry of Education of China, Shanghai JiaoTong University School of Medicine, Shanghai, 200025, China.

出版信息

Front Med China. 2010 Dec;4(4):363-70. doi: 10.1007/s11684-010-0210-7. Epub 2010 Nov 19.

Abstract

Chemical biology, using small molecules as probes to study the cellular signaling network, has developed rapidly in recent years. The interaction between chemistry and biology not only provides new insight into the understanding of cellular activities, but also generates new lead compounds for the treatment of diseases. Transcription factors and kinases such as retinoic acid receptor-alpha (RARα), acute myeloid leukemia 1 (AML1), CAAT/enhancer-binding protein α (C/EBPα), c-myc, and c-abl play important roles in the differentiation of hematopoietic stem/progenitor cells. Abnormalities in these proteins may cause the dysregulation of hematopoiesis and even the occurrence of leukemia. Ubiquitin-mediated protein degradation represents a critical mechanism in regulating the cellular levels and functions of these proteins. Thus, targeting protein degradation has been emerging as an important strategy to conquer malignant diseases. In this review, we will summarize the recent advances in the understanding of the roles of protein degradation in leukemia, with an emphasis on the mechanisms revealed by small molecules.

摘要

化学生物学利用小分子作为探针来研究细胞信号网络,近年来发展迅速。化学与生物学之间的相互作用不仅为理解细胞活动提供了新的视角,还产生了用于治疗疾病的新先导化合物。转录因子和激酶,如维甲酸受体α(RARα)、急性髓系白血病1(AML1)、CAAT/增强子结合蛋白α(C/EBPα)、c-myc和c-abl,在造血干/祖细胞的分化中起重要作用。这些蛋白质的异常可能导致造血功能失调,甚至引发白血病。泛素介导的蛋白质降解是调节这些蛋白质的细胞水平和功能的关键机制。因此,靶向蛋白质降解已成为攻克恶性疾病的重要策略。在本综述中,我们将总结近年来在理解蛋白质降解在白血病中的作用方面取得的进展,重点是小分子揭示的机制。

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