Prince Phillip Dental Hospital, The University of Honk Kong, Hong Kong SAR, China.
J Clin Periodontol. 2011 Mar;38 Suppl 11:178-81. doi: 10.1111/j.1600-051X.2010.01674.x.
Peri-implant diseases present in two forms - peri-implant mucositis and peri-implantitis.
The literature was systematically searched and critically reviewed. Four manuscripts were produced in specific topics identified as key areas to understand the microbial aetiology and the pathogenesis of peri-implant diseases and how the implant surface structure may affect pathogenesis.
While peri-implant mucositis represents the host response of the peri-implant tissues to the bacterial challenge that is not fundamentally different from gingivitis representing the host response to the bacterial challenge in the gingiva, peri-implantitis may differ from periodontitis both in the extent and the composition of cells in the lesion as well as the progression rate. A self-limiting process with a "protective" connective tissue capsule developing appears to dominate the periodontitis lesion while such a process may occasionally be lacking in peri-implantitis lesions. Bacterial biofilm formation on implant surfaces does not differ from that on tooth surfaces, but may be influenced by surface roughness. Nevertheless there is no evidence that such differences may influence the development of peri-implantitis.
It was agreed that clinical and radiographic data should routinely be obtained after prosthesis installation on implants in order to establish a baseline for the diagnosis of peri-implantitis during maintenance of implant patients.
种植体周围疾病有两种形式——种植体周围黏膜炎和种植体周围炎。
系统地搜索和批判性地回顾了文献。在确定为理解种植体周围疾病的微生物病因学和发病机制以及种植体表面结构如何影响发病机制的关键领域,产生了四份专题论文。
虽然种植体周围黏膜炎代表了种植体周围组织对细菌挑战的宿主反应,与代表牙龈对细菌挑战的宿主反应的牙龈炎并无根本不同,但种植体周围炎在病变中的细胞范围和组成以及进展速度方面可能与牙周炎不同。在牙周炎病变中,似乎主导着一种自限性过程,即形成具有“保护”作用的结缔组织囊,但这种过程在种植体周围炎病变中偶尔可能缺乏。种植体表面的细菌生物膜形成与牙面的生物膜形成没有区别,但可能受到表面粗糙度的影响。然而,没有证据表明这种差异可能影响种植体周围炎的发展。
大家一致认为,在种植体修复体安装后,应常规获得临床和影像学数据,以便在种植体患者的维护期间建立种植体周围炎的诊断基线。