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白细胞介素-1可诱导隐性营养不良型大疱性表皮松解症成纤维细胞产生胶原酶。

Interleukin-1 induces collagenase production by recessive dystrophic epidermolysis bullosa fibroblasts.

作者信息

Nomura K, Imaizumi T, Mikami H, Tamai K, Takahashi M, Hashimoto I

机构信息

Department of Dermatology, Hirosaki University School of Medicine, Japan.

出版信息

Arch Dermatol Res. 1990;282(6):379-82. doi: 10.1007/BF00372088.

Abstract

We examined the direct effect of interleukin-1 (IL-1) on the collagenase production by epidermolysis bullosa (EB) fibroblasts. Addition of IL-1 at concentrations of 2.5 x 10(-4) units/ml or below in the culture media greatly enhanced collagenase production by two cell lines of recessive dystrophic EB (RDEB) fibroblasts. They produced 4.82 +/- 0.04 to 5.93 +/- 0.39 units/ml of enzyme, as compared to 0.02 +/- 0.07 units/ml in the absence of IL-1. In contrast, collagenase production by two cell lines of dominant dystrophic EB (DDEB) and normal fibroblasts was not, or only slightly, increased up to 0.69 +/- 0.28 units/ml. IL-1 concentrations of 2.5 x 10(-3) units/ml or higher failed to induce collagenase production by all fibroblasts. 3H-thymidine uptake increased by about 110-376% of control after IL-1 treatment. In addition, these data were obtained using fibroblasts of the 13-15 passages, suggesting that the property might be determined genetically. Although RDEB seems to be a wide heterogeneous group, the present data strongly suggest that the property may be specific to and characteristic of some types of RDEB cells.

摘要

我们研究了白细胞介素 -1(IL -1)对大疱性表皮松解症(EB)成纤维细胞产生胶原酶的直接影响。在培养基中添加浓度为2.5×10⁻⁴单位/毫升或更低的IL -1,可显著增强两种隐性营养不良型EB(RDEB)成纤维细胞系的胶原酶产生。它们产生的酶量为4.82±0.04至5.93±0.39单位/毫升,而在无IL -1时为0.02±0.07单位/毫升。相比之下,两种显性营养不良型EB(DDEB)成纤维细胞系和正常成纤维细胞产生的胶原酶量未增加,或仅略有增加,最高达到0.69±0.28单位/毫升。浓度为2.5×10⁻³单位/毫升或更高的IL -1未能诱导所有成纤维细胞产生胶原酶。IL -1处理后,³H - 胸腺嘧啶核苷摄取量比对照增加了约110 - 376%。此外,这些数据是使用第13 - 15代的成纤维细胞获得的,表明该特性可能由基因决定。尽管RDEB似乎是一个广泛的异质群体,但目前的数据强烈表明该特性可能是某些类型的RDEB细胞所特有的特征。

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