相似文献
1
Calcium-dependent signaling in Dupuytren's disease.掌腱膜挛缩症中的钙依赖性信号传导
Hand (N Y). 2011 Jun;6(2):159-64. doi: 10.1007/s11552-010-9314-4. Epub 2010 Dec 15.
2
Dupuytren's fibroblast contractility by sphingosine-1-phosphate is mediated through non-muscle myosin II.鞘氨醇-1-磷酸对杜普伊特伦挛缩症成纤维细胞收缩性的作用是通过非肌肉肌球蛋白II介导的。
J Hand Surg Am. 2010 Oct;35(10):1580-8. doi: 10.1016/j.jhsa.2010.07.009.
3
Calmodulin-myosin light chain kinase inhibition changes fibroblast-populated collagen lattice contraction, cell migration, focal adhesion formation, and wound contraction.钙调蛋白-肌球蛋白轻链激酶抑制作用会改变成纤维细胞填充的胶原晶格收缩、细胞迁移、粘着斑形成和伤口收缩。
Wound Repair Regen. 2004 Sep-Oct;12(5):505-11. doi: 10.1111/j.1067-1927.2004.012502.x.
4
Dupuytren's disease susceptibility gene, EPDR1, is involved in myofibroblast contractility.掌腱膜挛缩症易感基因EPDR1参与肌成纤维细胞收缩。
J Dermatol Sci. 2016 Aug;83(2):131-7. doi: 10.1016/j.jdermsci.2016.04.015. Epub 2016 May 7.
5
Correlation of alpha-smooth muscle actin expression and contraction in Dupuytren's disease fibroblasts.杜普伊特伦挛缩病成纤维细胞中α-平滑肌肌动蛋白表达与收缩的相关性
J Hand Surg Am. 1995 May;20(3):450-5. doi: 10.1016/S0363-5023(05)80105-4.
6
Tamoxifen decreases fibroblast function and downregulates TGF(beta2) in dupuytren's affected palmar fascia.他莫昔芬可降低掌腱膜挛缩症患者掌腱膜中纤维母细胞的功能，并下调转化生长因子β2（TGF-β2）。
J Surg Res. 2002 Apr;103(2):146-52. doi: 10.1006/jsre.2001.6350.
7
Pharmacologic regulation of Dupuytren's fibroblast contraction in vitro.体外掌腱膜成纤维细胞收缩的药理学调控
J Hand Surg Am. 1996 Nov;21(6):1065-70. doi: 10.1016/S0363-5023(96)80317-0.
8
Generation of contractile force by cultured Dupuytren's disease and normal palmar fibroblasts.培养的掌腱膜挛缩症和正常掌部成纤维细胞产生收缩力的情况。
Tissue Cell. 1994 Oct;26(5):747-56. doi: 10.1016/0040-8166(94)90057-4.
9
Post-transcriptional regulation of alpha-smooth muscle actin determines the contractile phenotype of Dupuytren's nodular cells.转录后调控α-平滑肌肌动蛋白决定了迪普屈恩结节细胞的收缩表型。
J Cell Physiol. 2010 Sep;224(3):681-90. doi: 10.1002/jcp.22167.
10
An insight into Dupuytren's contracture.深入了解杜普伊特伦挛缩症。
Ann R Coll Surg Engl. 1992 May;74(3):156-60; discussion 161.
引用本文的文献
1
Comparison of Rheological Properties of Healthy versus Dupuytren Fibroblasts When Treated with a Cell Contraction Inhibitor by Atomic Force Microscope.原子力显微镜下细胞收缩抑制剂处理的健康成纤维细胞与杜普伊特伦挛缩症成纤维细胞流变特性比较。
Int J Mol Sci. 2023 Jan 20;24(3):2043. doi: 10.3390/ijms24032043.
2
The Role of Calmodulin in Tumor Cell Migration, Invasiveness, and Metastasis.钙调蛋白在肿瘤细胞迁移、侵袭和转移中的作用。
Int J Mol Sci. 2020 Jan 24;21(3):765. doi: 10.3390/ijms21030765.
本文引用的文献
1
Antifibrotic medication using a combination of N-acetyl-L-cystein (NAC) and ACE inhibitors can prevent the recurrence of Dupuytren's disease.使用 N-乙酰-L-半胱氨酸 (NAC) 和血管紧张素转换酶抑制剂的抗纤维化药物可以预防掌腱膜挛缩症的复发。
Med Hypotheses. 2009 Nov;73(5):659-61. doi: 10.1016/j.mehy.2009.08.011. Epub 2009 Sep 1.
2
Screening of potential prodrugs on cells derived from Dupuytren's disease patients.对来自掌腱膜挛缩症患者的细胞进行潜在前药的筛选。
Biomed Pharmacother. 2009 Sep;63(8):577-85. doi: 10.1016/j.biopha.2008.10.005. Epub 2008 Oct 31.
3
Matrix metalloproteinase inhibition reduces contraction by dupuytren fibroblasts.基质金属蛋白酶抑制可减少杜普伊特伦挛缩症成纤维细胞的收缩。
J Hand Surg Am. 2008 Nov;33(9):1608-16. doi: 10.1016/j.jhsa.2008.06.013.
4
Mechanisms of actin stress fibre assembly.肌动蛋白应力纤维组装的机制。
J Microsc. 2008 Sep;231(3):446-54. doi: 10.1111/j.1365-2818.2008.02057.x.
5
The natural history of Peyronie's disease.
Arch Esp Urol. 2007 Apr;60(3):326-31. doi: 10.4321/s0004-06142007000300021.
6
Dupuytren's disease: anatomy, pathology, presentation, and treatment.
Instr Course Lect. 2007;56:101-11.
7
Dupuytren's diathesis revisited: Evaluation of prognostic indicators for risk of disease recurrence.再探杜普伊特伦氏素质：疾病复发风险的预后指标评估
J Hand Surg Am. 2006 Dec;31(10):1626-34. doi: 10.1016/j.jhsa.2006.09.006.
8
Rho kinase differentially regulates phosphorylation of nonmuscle myosin II isoforms A and B during cell rounding and migration.在细胞变圆和迁移过程中，Rho激酶对非肌肉肌球蛋白II亚型A和B的磷酸化具有不同的调节作用。
J Biol Chem. 2006 Nov 24;281(47):35873-83. doi: 10.1074/jbc.M605343200. Epub 2006 Oct 3.
9
The heritability of Dupuytren's disease: familial aggregation and its clinical significance.掌腱膜挛缩症的遗传度：家族聚集性及其临床意义。
J Hand Surg Am. 2006 Feb;31(2):204-10. doi: 10.1016/j.jhsa.2005.09.018.
10
Calmodulin-myosin light chain kinase inhibition changes fibroblast-populated collagen lattice contraction, cell migration, focal adhesion formation, and wound contraction.钙调蛋白-肌球蛋白轻链激酶抑制作用会改变成纤维细胞填充的胶原晶格收缩、细胞迁移、粘着斑形成和伤口收缩。
Wound Repair Regen. 2004 Sep-Oct;12(5):505-11. doi: 10.1111/j.1067-1927.2004.012502.x.
Zotero 插件