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本文引用的文献

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Conceptual framework for cutting the pancreatic cancer fuel supply.
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Heterogeneity and targeting of pancreatic cancer stem cells.
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The pancreas cancer microenvironment.
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ATM mutations in patients with hereditary pancreatic cancer.
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Presence of somatic mutations in most early-stage pancreatic intraepithelial neoplasia.
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Whole-exome sequencing of neoplastic cysts of the pancreas reveals recurrent mutations in components of ubiquitin-dependent pathways.
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Recurrent GNAS mutations define an unexpected pathway for pancreatic cyst development.
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Altered telomeres in tumors with ATRX and DAXX mutations.
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Everolimus for advanced pancreatic neuroendocrine tumors.
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