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Switching from protease-independent to protease-dependent cancer cell invasion.

作者信息

He Lijuan, Wirtz Denis

机构信息

Department of Chemical and Biomolecular Engineering, The Johns Hopkins University, Baltimore, Maryland; Johns Hopkins Physical Sciences-Oncology Center, The Johns Hopkins University, Baltimore, Maryland.

Department of Chemical and Biomolecular Engineering, The Johns Hopkins University, Baltimore, Maryland; Johns Hopkins Physical Sciences-Oncology Center, The Johns Hopkins University, Baltimore, Maryland; Departments of Oncology and Pathology and Sidney Kimmel Comprehensive Cancer Center, The Johns Hopkins University, Baltimore, Maryland.

出版信息

Biophys J. 2014 Dec 2;107(11):2484-5. doi: 10.1016/j.bpj.2014.10.047.

Abstract
摘要

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本文引用的文献

1
3D traction stresses activate protease-dependent invasion of cancer cells.
Biophys J. 2014 Dec 2;107(11):2528-37. doi: 10.1016/j.bpj.2014.07.078.
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Cellular traction stresses mediate extracellular matrix degradation by invadopodia.
Acta Biomater. 2014 May;10(5):1886-96. doi: 10.1016/j.actbio.2013.12.058. Epub 2014 Jan 8.
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The Arp2/3 complex mediates multigeneration dendritic protrusions for efficient 3-dimensional cancer cell migration.
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Mechanical modes of 'amoeboid' cell migration.
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