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一类参与选择性自噬和多聚谷氨酰胺蛋白清除的新型泛素-Atg8受体。

A new class of ubiquitin-Atg8 receptors involved in selective autophagy and polyQ protein clearance.

作者信息

Lu Kefeng, Psakhye Ivan, Jentsch Stefan

机构信息

a Department of Molecular Cell Biology; Max Planck Institute of Biochemistry ; Martinsried , Germany.

出版信息

Autophagy. 2014;10(12):2381-2. doi: 10.4161/15548627.2014.981919.

Abstract

Selective ubiquitin-dependent autophagy mediates the disposal of superfluous cellular structures and clears cells of protein aggregates such as polyQ proteins linked to Huntington disease. Crucial selectivity factors of this pathway are ubiquitin-Atg8 receptors such as human SQSTM1/p62, which recognize ubiquitinated cargoes and deliver them to phagophores for degradation. Contrasting previous beliefs, we recently showed that ubiquitin-dependent autophagy is not restricted to higher eukaryotes but also exists in yeast with Cue5, harboring a ubiquitin-binding CUE domain, being a ubiquitin-Atg8 receptor. Notably, the human CUE domain protein TOLLIP is functionally similar to yeast Cue5, indicating that Cue5/TOLLIP (CUET) proteins represent a new and conserved class of autophagy receptors. Remarkably, both Cue5 in yeast and TOLLIP in human cells mediate efficient clearance of aggregation-prone polyQ proteins derived from human HTT/huntingtin. Indeed, TOLLIP is potentially more potent in polyQ clearance than SQSTM1/p62 in HeLa cells, suggesting that TOLLIP, also implicated in innate immunity, may be significant for human health and disease.

摘要

选择性泛素依赖性自噬介导多余细胞结构的清除,并清除细胞中的蛋白质聚集体,如与亨廷顿病相关的多聚谷氨酰胺蛋白。该途径的关键选择性因子是泛素-自噬相关蛋白8(Atg8)受体,如人类的SQSTM1/p62,它识别泛素化的货物并将其递送至吞噬体进行降解。与之前的观点不同,我们最近发现泛素依赖性自噬并不局限于高等真核生物,在酵母中也存在,其中含有泛素结合CUE结构域的Cue5作为泛素-Atg8受体。值得注意的是,人类CUE结构域蛋白Toll相互作用蛋白(TOLLIP)在功能上与酵母Cue5相似,这表明Cue5/TOLLIP(CUET)蛋白代表了一类新的保守的自噬受体。值得注意的是,酵母中的Cue5和人类细胞中的TOLLIP都能有效清除源自人类亨廷顿蛋白(HTT/huntingtin)的易聚集多聚谷氨酰胺蛋白。事实上,在HeLa细胞中,TOLLIP在清除多聚谷氨酰胺方面可能比SQSTM1/p62更有效,这表明同样参与先天免疫的TOLLIP可能对人类健康和疾病具有重要意义。

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