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9至18岁反社会人格的发展:基因与环境

Psychopathic personality development from ages 9 to 18: Genes and environment.

作者信息

Tuvblad Catherine, Wang Pan, Bezdjian Serena, Raine Adrian, Baker Laura A

机构信息

University of Southern California.

University of Pennsylvania.

出版信息

Dev Psychopathol. 2016 Feb;28(1):27-44. doi: 10.1017/S0954579415000267. Epub 2015 May 5.

Abstract

The genetic and environmental etiology of individual differences was examined in initial level and change in psychopathic personality from ages 9 to 18 years. A piecewise growth curve model, in which the first change score (G1) influenced all ages (9-10, 11-13, 14-15, and 16-18 years) and the second change score (G2) only influenced ages 14-15 and 16-18 years, fit the data better did than the standard single slope model, suggesting a turning point from childhood to adolescence. The results indicated that variations in levels and both change scores were mainly due to genetic (A) and nonshared environmental (E) influences (i.e., AE structure for G0, G1, and G2). No sex differences were found except on the mean values of level and change scores. Based on caregiver ratings, about 81% of variance in G0, 89% of variance in G1, and 94% of variance in G2 were explained by genetic factors, whereas for youth self-reports, these three proportions were 94%, 71%, and 66%, respectively. The larger contribution of genetic variance and covariance in caregiver ratings than in youth self-reports may suggest that caregivers considered the changes in their children to be more similar as compared to how the children viewed themselves.

摘要

研究了9至18岁个体差异的遗传和环境病因,以及精神病态人格的初始水平和变化情况。一个分段生长曲线模型,其中第一个变化分数(G1)影响所有年龄段(9至10岁、11至13岁、14至15岁和16至18岁),第二个变化分数(G2)仅影响14至15岁和16至18岁,比标准的单斜率模型更适合数据,这表明从童年到青春期存在一个转折点。结果表明,水平和两个变化分数的差异主要归因于遗传(A)和非共享环境(E)的影响(即G0、G1和G2的AE结构)。除了水平和变化分数的平均值外,未发现性别差异。根据照顾者的评分,遗传因素解释了G0中约81%的方差、G1中89%的方差和G2中94%的方差;而对于青少年的自我报告,这三个比例分别为94%、71%和66%。遗传方差和协方差在照顾者评分中的贡献大于在青少年自我报告中的贡献,这可能表明照顾者认为他们孩子的变化比孩子自己认为的更相似。

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