Thompson A, Illescas F F, Chiu R C
Montreal General Hospital, Quebec, Canada.
Ann Thorac Surg. 1989 Feb;47(2):247-9. doi: 10.1016/0003-4975(89)90279-8.
Traumatic asphyxia secondary to a crush injury of the chest is characterized by craniocervical cyanosis, subconjunctival hemorrhage, and severe vascular engorgement of the head and neck. These signs are believed to be due to high venous pressures causing stasis and capillary rupture. A "fear response" that produces a strong Valsalva maneuver is thought to be necessary for their development. The lower torso seems to be protected, and previously this was thought to be due to its superior system of valves. We present here ultrasonographic evidence that the inferior vena cava is compressed or obliterated during a Valsalva maneuver, and propose that this compression protects the lower torso during traumatic asphyxia.
胸部挤压伤继发的创伤性窒息表现为颅颈青紫、结膜下出血以及头颈部严重的血管充血。这些体征被认为是由于静脉压升高导致血液淤滞和毛细血管破裂所致。一种产生强烈瓦尔萨尔瓦动作的“恐惧反应”被认为是这些体征出现所必需的。下半身似乎受到保护,以前认为这是由于其优越的瓣膜系统。我们在此展示超声证据,即瓦尔萨尔瓦动作时下腔静脉会被压缩或闭塞,并提出这种压缩在创伤性窒息时保护了下半身。