Reply to Vicetti Miguel et al., "Setting Sights on Chlamydia Immunity's Central Paradigm: Can We Hit a Moving Target?".
作者信息
Johnson Raymond M, Brunham Robert C
机构信息
Section of Infectious Diseases, Yale University School of Medicine, New Haven, Connecticut, USA.
Vaccine Research Laboratory, University of British Columbia Centre for Disease Control, Vancouver, British Columbia, Canada
出版信息
Infect Immun. 2017 Jun 20;85(7). doi: 10.1128/IAI.00223-17. Print 2017 Jul.
Abstract
摘要
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Human CD8+ T-cells recognizing peptides from Mycobacterium tuberculosis (Mtb) presented by HLA-E have an unorthodox Th2-like, multifunctional, Mtb inhibitory phenotype and represent a novel human T-cell subset.识别由HLA-E呈递的来自结核分枝杆菌（Mtb）肽段的人类CD8 + T细胞具有非传统的Th2样多功能Mtb抑制表型，代表了一种新型人类T细胞亚群。
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Respiratory synctial virus infection in BALB/c mice previously immunized with formalin-inactivated virus induces enhanced pulmonary inflammatory response with a predominant Th2-like cytokine pattern.用福尔马林灭活病毒预先免疫的BALB/c小鼠感染呼吸道合胞病毒后，会诱导出以Th2样细胞因子模式为主的增强型肺部炎症反应。
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Respiratory syncytial virus disease in infants despite prior administration of antigenic inactivated vaccine.尽管之前接种了抗原性灭活疫苗，但婴儿仍患呼吸道合胞病毒病。
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