Is multiple system atrophy an infectious disease?
作者信息
Wenning Gregor, Trojanowski John Q, Kaufmann Horacio, Wisniewski Thomas, Rocca Walter A, Low Phillip A
机构信息
Department of Neurology, Innsbruck Medical University, Innsbruck, Austria.
Department of Pathology and Laboratory Medicine, Perelman School of Medicine at the University of Pennsylvania, Philadelphia, PA.
出版信息
Ann Neurol. 2018 Jan;83(1):10-12. doi: 10.1002/ana.25132. Epub 2018 Jan 14.
Abstract
摘要
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2
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3
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4
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5
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8
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10
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