Anderson Robert H, Spicer Diane E, Mori Shumpei
Institute of Genetic Medicine, Newcastle University, Newcastle-upon-Tyne NE1 3BZ, UK.
Johns Hopkins All Children's Heart Institute, St. Petersburg, FL 33701, USA.
J Cardiovasc Dev Dis. 2016 Apr 19;3(2):17. doi: 10.3390/jcdd3020017.
In the review, now published as part of the special issue devoted to the development of the conduction tissues, de Vries and his colleagues discuss the potential role of the so-called "dead-end tract" as a substrate for arrhythmogenesis [1].[...].
在现已作为关于传导组织发育的特刊一部分发表的这篇综述中,德弗里斯及其同事讨论了所谓“终末束”作为心律失常发生基质的潜在作用[1]。[...]