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PRMT5 对于在膝关节和长骨中形成不同的软骨特征是必要的。

PRMT5 is necessary to form distinct cartilage identities in the knee and long bone.

机构信息

Department of Molecular Biosciences, University of Texas at Austin, 100 E 24th Street, Stop A5000, Austin, TX, 78712, USA.

Department of Pediatrics, Dell Pediatrics Research Institute, University of Texas at Austin Dell Medical School, 1400 Barbara Jordan Blvd, Austin, TX, 78723, USA.

出版信息

Dev Biol. 2019 Dec 15;456(2):154-163. doi: 10.1016/j.ydbio.2019.08.012. Epub 2019 Aug 20.

Abstract

During skeletal development, limb progenitors become specified as chondrocytes and subsequently differentiate into specialized cartilage compartments. We previously showed that the arginine dimethyl transferase, PRMT5, is essential for regulating the specification of progenitor cells into chondrocytes within early limb buds. Here, we report that PRMT5 regulates the survival of a separate progenitor domain that gives rise to the patella. Independent of its role in knee development, PRMT5 regulates several distinct types of chondrocyte differentiation within the long bones. Chondrocytes lacking PRMT5 have a striking blockage in hypertrophic chondrocyte differentiation and are marked by abnormal gene expression. PRMT5 remains important for articular cartilage and hypertrophic cell identity during adult stages, indicating an ongoing role in homeostasis of these tissues. We conclude that PRMT5 is required for distinct steps of early and late chondrogenic specialization and is thus a critical component of multiple aspects of long bone development and maintenance.

摘要

在骨骼发育过程中,肢体祖细胞被特化为软骨细胞,随后进一步分化为特定的软骨区域。我们之前的研究表明,精氨酸二甲基转移酶 PRMT5 对于调节早期肢芽中的祖细胞向软骨细胞的特化具有重要作用。在这里,我们报告 PRMT5 调节了髌骨前体的另一个独立祖细胞区域的存活。PRMT5 独立于其在膝关节发育中的作用,还调节长骨中几种不同类型的软骨细胞分化。缺乏 PRMT5 的软骨细胞在肥大软骨细胞分化中出现明显的阻滞,并表现出异常的基因表达。PRMT5 在成年阶段对于关节软骨和肥大细胞的特征仍然很重要,表明其在这些组织的稳态中发挥持续作用。我们得出结论,PRMT5 对于早期和晚期软骨细胞特化的不同步骤都是必需的,因此是长骨发育和维持的多个方面的关键组成部分。

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