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The Intricate Web of Phospholipase As and Specific Features of Airway Hyperresponsiveness in Asthma.

作者信息

Murphy Ryan C, Altemeier William A, Lai Ying, Hallstrand Teal S

机构信息

University of Washington, Seattle, Washington.

出版信息

Am J Respir Cell Mol Biol. 2020 Oct;63(4):543-545. doi: 10.1165/rcmb.2020-0131LE.

DOI:10.1165/rcmb.2020-0131LE
PMID:32484733
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC7528919/
Abstract
摘要

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Reply to: The Intricate Web of Phospholipase As and Specific Features of Airway Hyperresponsiveness in Asthma.回复：磷脂酶A与哮喘气道高反应性的复杂关系及特异性特征
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Exercise-induced alterations in phospholipid hydrolysis, airway surfactant, and eicosanoids and their role in airway hyperresponsiveness in asthma.运动诱导的磷脂水解、气道表面活性剂和类二十烷酸的改变及其在哮喘气道高反应性中的作用。
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本文引用的文献

1
Asthma: An Untoward Consequence of Endurance Sports?哮喘：耐力运动的不良后果？
Am J Respir Cell Mol Biol. 2020 Jul;63(1):7-8. doi: 10.1165/rcmb.2020-0092ED.
2
Cysteinyl Leukotriene Synthesis via Phospholipase A2 Group IV Mediates Exercise-induced Bronchoconstriction and Airway Remodeling.通过磷脂酶 A2 组 IV 介导半胱氨酰白三烯合成介导运动诱导的支气管收缩和气道重塑。
Am J Respir Cell Mol Biol. 2020 Jul;63(1):57-66. doi: 10.1165/rcmb.2019-0325OC.
3
Airway brush cells generate cysteinyl leukotrienes through the ATP sensor P2Y2.气道刷状细胞通过 ATP 感受器 P2Y2 产生半胱氨酰白三烯。
Sci Immunol. 2020 Jan 17;5(43). doi: 10.1126/sciimmunol.aax7224.
4
Airway epithelium-shifted mast cell infiltration regulates asthmatic inflammation via IL-33 signaling.气道上皮细胞迁移的肥大细胞浸润通过 IL-33 信号调节哮喘炎症。
J Clin Invest. 2019 Nov 1;129(11):4979-4991. doi: 10.1172/JCI126402.
5
ERS technical standard on bronchial challenge testing: pathophysiology and methodology of indirect airway challenge testing.ERS 技术标准：支气管激发试验——间接气道激发试验的病理生理学和方法学。
Eur Respir J. 2018 Nov 15;52(5). doi: 10.1183/13993003.01033-2018. Print 2018 Nov.
6
Resolvins in inflammation: emergence of the pro-resolving superfamily of mediators.解决素在炎症反应中的作用：促解决介质的内源性超家族的出现。
J Clin Invest. 2018 Jul 2;128(7):2657-2669. doi: 10.1172/JCI97943. Epub 2018 May 14.
7
Macrophages regulate lung ILC2 activation via Pla2g5-dependent mechanisms.巨噬细胞通过 Pla2g5 依赖的机制调节肺 ILC2 的激活。
Mucosal Immunol. 2018 May;11(3):615-626. doi: 10.1038/mi.2017.99. Epub 2017 Dec 20.
8
ERS technical standard on bronchial challenge testing: general considerations and performance of methacholine challenge tests.ERS 技术标准：支气管激发试验——一般考虑和乙酰甲胆碱激发试验的实施。
Eur Respir J. 2017 May 1;49(5). doi: 10.1183/13993003.01526-2016. Print 2017 May.
9
Increased density of intraepithelial mast cells in patients with exercise-induced bronchoconstriction regulated through epithelially derived thymic stromal lymphopoietin and IL-33.运动诱发性支气管收缩患者上皮内肥大细胞密度增加，其通过上皮来源的胸腺基质淋巴细胞生成素和白细胞介素-33进行调节。
J Allergy Clin Immunol. 2014 May;133(5):1448-55. doi: 10.1016/j.jaci.2013.08.052. Epub 2013 Nov 9.
10
Group V secretory phospholipase A2 is involved in macrophage activation and is sufficient for macrophage effector functions in allergic pulmonary inflammation.组 V 分泌型磷脂酶 A2 参与巨噬细胞激活，并足以在变应性肺部炎症中发挥巨噬细胞效应功能。
J Immunol. 2013 Jun 15;190(12):5927-38. doi: 10.4049/jimmunol.1203202. Epub 2013 May 6.