主动脉内皮损伤后的补体激活。
Complement activation after aortic endothelial injury.
作者信息
Seifert P S, Catalfamo J L, Dodds J
出版信息
Semin Thromb Hemost. 1986 Oct;12(4):280-3. doi: 10.1055/s-2007-1003567.
PMID:3491429
Abstract
Ischemic endothelial cell injury results in C activation that precedes partially via the classic pathway. This may be relevant to clinical situations of ischemia such as occur during embolization or thrombus formation. The generation of C activation products (such as C3a and C5a) may mediate many of the inflammatory changes observed in infarcted tissues.
摘要
缺血性内皮细胞损伤导致补体（C）激活，部分是通过经典途径启动的。这可能与缺血的临床情况相关，比如在栓塞或血栓形成过程中发生的缺血。补体激活产物（如C3a和C5a）的产生可能介导了在梗死组织中观察到的许多炎症变化。
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