Stoica E, Enulescu O
Funct Neurol. 1986 Jul-Sep;1(3):261-8.
Hypertensives with (25 cases) or without (27 cases) cerebral hemorrhage (CH) and normotensives (25 cases) were exposed from 7 to 8.30 a.m. to a low light input and from 8.30 to 10 to a high light input (HLI), and urinary epinephrine (E) and norepinephrine (NE) were measured at the end of these periods. In normotensives HLI had no effect on E and NE excretion; in hypertensives with and without CH it increased E excretion and depressed NE excretion. Light activation of E release may be responsible for the daytime occurrence of CH in hypertensives.
患有脑出血（CH）的高血压患者（25例）和未患脑出血的高血压患者（27例）以及血压正常者（25例），于上午7点至8点30分暴露于低光照强度下，8点30分至10点暴露于高光照强度（HLI）下，在这两个时间段结束时测量尿肾上腺素（E）和去甲肾上腺素（NE）。在血压正常者中，高光照强度对E和NE的排泄没有影响；在患有和未患脑出血的高血压患者中，高光照强度增加了E的排泄并降低了NE的排泄。E释放的光激活可能是高血压患者白天发生脑出血的原因。
