Wu Zhihong, Tang Jianjun, Zhu Qingyi, Hu Lin, Liu Zhenjiang, Li Xuping, Liu Qiming, Zhou Shenghua, Chen Mingxian
Department of Cardiology, The Second Xiangya Hospital of Central South University, Changsha, China.
Front Cardiovasc Med. 2022 Dec 6;9:993903. doi: 10.3389/fcvm.2022.993903. eCollection 2022.
A 71-year-old female with a dual-chamber pacemaker presented to our hospital complaining of repeated chest pain. She was diagnosed with unstable angina. On day 7, the patient suddenly suffered cardiopulmonary arrest due to an inferior ST segment elevation myocardial infarction (STEMI). Pacemaker lost capture was suspected and was later confirmed by a pacemaker check with a high pacing threshold and a low sensing parameter. Emergency coronary angiography revealed that a large filling defect remained due to an extensive thrombus in the proximal left circumflex (LCX) with thrombolysis in myocardial infarction (TIMI) grade 2 flow, and then a repeat thrombus aspiration was performed. After reperfusion, the parameters of the right ventricular lead were gradually returned. We conclude that the loss of the right ventricular lead pacing occurred in this case of acute coronary syndrome (ACS) induced by an LCX thrombus due to an LCX supplying the right ventricular septal.
一名71岁植入双腔起搏器的女性因反复胸痛前来我院就诊。她被诊断为不稳定型心绞痛。在第7天,患者因下壁ST段抬高型心肌梗死(STEMI)突然发生心肺骤停。怀疑起搏器失夺获,随后通过起搏器检查证实,起搏阈值高且感知参数低。急诊冠状动脉造影显示,由于左旋支(LCX)近端广泛血栓形成,TIMI血流2级,仍有较大充盈缺损,随后进行了再次血栓抽吸。再灌注后,右心室导线参数逐渐恢复。我们得出结论,在本例由LCX血栓引起的急性冠状动脉综合征(ACS)中,右心室导线起搏丧失是由于LCX供应右心室间隔所致。