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Glua1 Ubiquitination in Synaptic Plasticity and Cognitive Functions: Implications for Neurodegeneration.

作者信息

Hiskens Matthew I

机构信息

Mackay Institute of Research and Innovation, Mackay Base Hospital, Mackay, Queensland 4740, Australia

出版信息

J Neurosci. 2024 Jan 17;44(3):e2018232024. doi: 10.1523/JNEUROSCI.2018-23.2024.

Abstract
摘要

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本文引用的文献

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Ubiquitination of the GluA1 Subunit of AMPA Receptors Is Required for Synaptic Plasticity, Memory, and Cognitive Flexibility.
J Neurosci. 2023 Jul 26;43(30):5448-5457. doi: 10.1523/JNEUROSCI.1542-22.2023. Epub 2023 Jul 7.
2
The Emerging Roles of E3 Ligases and DUBs in Neurodegenerative Diseases.
Mol Neurobiol. 2023 Jan;60(1):247-263. doi: 10.1007/s12035-022-03063-3. Epub 2022 Oct 19.
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Historical perspective and progress on protein ubiquitination at glutamatergic synapses.
Neuropharmacology. 2021 Sep 15;196:108690. doi: 10.1016/j.neuropharm.2021.108690. Epub 2021 Jun 29.
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Neurosci Lett. 2019 Jun 11;703:68-78. doi: 10.1016/j.neulet.2019.03.016. Epub 2019 Mar 16.
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The AMPA Receptor Code of Synaptic Plasticity.
Neuron. 2018 Oct 24;100(2):314-329. doi: 10.1016/j.neuron.2018.10.018.
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The Decrease of Uch-L1 Activity Is a Common Mechanism Responsible for Aβ 42 Accumulation in Alzheimer's and Vascular Disease.
Front Aging Neurosci. 2017 Sep 29;9:320. doi: 10.3389/fnagi.2017.00320. eCollection 2017.
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