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成人先天性肌无力综合征:临床特征、诊断和长期预后。

Congenital myasthenic syndromes in adults: clinical features, diagnosis and long-term prognosis.

机构信息

Centre de référence des Maladies Neuromusculaires Nord/Est/Ile de France, Institut de Myologie, Hôpital Pitié-Salpêtrière, Assistance Publique des Hôpitaux de Paris, 75013 Paris, France.

Service d'ENMG et de pathologies neuromusculaires, centre de référence des maladies neuromusculaires PACA-Réunion-Rhône-Alpes, Hôpital Neurologique Pierre Wertheimer, Hospices Civils de Lyon, Groupement Est, 69500 Bron, France.

出版信息

Brain. 2024 Nov 4;147(11):3849-3862. doi: 10.1093/brain/awae124.

DOI:10.1093/brain/awae124
PMID:38696726
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC11531845/
Abstract

Congenital myasthenic syndromes (CMS) are clinically and genetically heterogeneous diseases caused by mutations affecting neuromuscular transmission. Even if the first symptoms mainly occur during childhood, adult neurologists must confront this challenging diagnosis and manage these patients throughout their adulthood. However, long-term follow-up data from large cohorts of CMS patients are lacking, and the long-term prognosis of these patients is largely unknown. We report the clinical features, diagnostic difficulties, and long-term prognosis of a French nationwide cohort of 235 adult patients with genetically confirmed CMS followed in 23 specialized neuromuscular centres. Data were retrospectively analysed. Of the 235 patients, 123 were female (52.3%). The diagnosis was made in adulthood in 139 patients, 110 of whom presented their first symptoms before the age of 18. Mean follow-up time between first symptoms and last visit was 34 years [standard deviation (SD) = 15.1]. Pathogenic variants were found in 19 disease-related genes. CHRNE-low expressor variants were the most common (23.8%), followed by variants in DOK7 (18.7%) and RAPSN (14%). Genotypes were clustered into four groups according to the initial presentation: ocular group (CHRNE-LE, CHRND, FCCMS), distal group (SCCMS), limb-girdle group (RAPSN, COLQ, DOK7, GMPPB, GFPT1), and a variable-phenotype group (MUSK, AGRN). The phenotypical features of CMS did not change throughout life. Only four genotypes had a proportion of patients requiring intensive care unit admission that exceeded 20%: RAPSN (54.8%), MUSK (50%), DOK7 (38.6%) and AGRN (25.0%). In RAPSN and MUSK patients most ICU admissions occurred before age 18 years and in DOK7 and AGRN patients at or after 18 years of age. Different patterns of disease course (stability, improvement and progressive worsening) may succeed one another in the same patient throughout life, particularly in AGRN, DOK7 and COLQ. At the last visit, 55% of SCCMS and 36.3% of DOK7 patients required ventilation; 36.3% of DOK7 patients, 25% of GMPPB patients and 20% of GFPT1 patients were wheelchair-bound; most of the patients who were both wheelchair-bound and ventilated were DOK7 patients. Six patients died in this cohort. The positive impact of therapy was striking, even in severely affected patients. In conclusion, even if motor and/or respiratory deterioration could occur in patients with initially moderate disease, particularly in DOK7, SCCMS and GFPT1 patients, the long-term prognosis for most CMS patients was favourable, with neither ventilation nor wheelchair needed at last visit. CHRNE-LE patients did not worsen during adulthood and RAPSN patients, often severely affected in early childhood, subsequently improved.

摘要

先天性肌无力综合征(CMS)是由影响神经肌肉传递的突变引起的临床和遗传异质性疾病。即使首发症状主要发生在儿童期,成年神经科医生也必须面对这一具有挑战性的诊断,并在成年期管理这些患者。然而,CMS 患者的大型队列的长期随访数据缺乏,这些患者的长期预后在很大程度上是未知的。我们报告了法国全国 235 名经基因证实的 CMS 成年患者的临床特征、诊断困难和长期预后,这些患者在 23 个专门的神经肌肉中心接受治疗。数据是回顾性分析的。在 235 名患者中,123 名女性(52.3%)。139 名患者在成年期诊断,其中 110 名患者在 18 岁之前出现首发症状。首发症状至最后一次就诊的平均随访时间为 34 年[标准差(SD)=15.1]。在 19 个疾病相关基因中发现了致病性变异。CHRNE 低表达变异体最为常见(23.8%),其次是 DOK7(18.7%)和 RAPSN(14%)。根据初始表现将基因型分为四组:眼肌组(CHRNE-LE、CHRND、FCCMS)、远端组(SCCMS)、肢体带组(RAPSN、COLQ、DOK7、GMPPB、GFPT1)和可变表型组(MUSK、AGRN)。CMS 的表型特征在整个生命周期中都没有改变。只有四种基因型需要入住重症监护病房的患者比例超过 20%:RAPSN(54.8%)、MUSK(50%)、DOK7(38.6%)和 AGRN(25.0%)。在 RAPSN 和 MUSK 患者中,大多数 ICU 入院发生在 18 岁之前,而在 DOK7 和 AGRN 患者中则发生在 18 岁或之后。在同一患者中,不同的疾病进程模式(稳定、改善和进行性恶化)可能会在一生中相继出现,特别是在 AGRN、DOK7 和 COLQ 中。在最后一次就诊时,55%的 SCCMS 和 36.3%的 DOK7 患者需要通气;36.3%的 DOK7 患者、25%的 GMPPB 患者和 20%的 GFPT1 患者需要坐轮椅;大多数同时需要坐轮椅和通气的患者都是 DOK7 患者。该队列中有 6 名患者死亡。治疗的积极影响是显著的,即使是在最初病情中等的患者中,特别是在 DOK7、SCCMS 和 GFPT1 患者中。大多数 CMS 患者的长期预后良好,最后一次就诊时无需通气或坐轮椅。CHRNE-LE 患者在成年期没有恶化,RAPSN 患者在儿童早期常受严重影响,随后有所改善。

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https://cdn.ncbi.nlm.nih.gov/pmc/blobs/d874/11531845/9f4362ba8588/awae124f1.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/d874/11531845/6a748435d775/awae124f2.jpg
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New mutation in the β1 propeller domain of LRP4 responsible for congenital myasthenic syndrome associated with Cenani-Lenz syndrome.
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