Kidwell Joseph Lewis
Independent Researcher, Willmar, MN, United States.
Front Psychol. 2025 Aug 5;16:1617192. doi: 10.3389/fpsyg.2025.1617192. eCollection 2025.
ADHD and Autism Spectrum Disorder (ASD) represent distinct neurodevelopmental conditions with unique profiles, yet they share susceptibility to environmental pressures that may exacerbate cognitive mismatches. This paper argues that Attention-Deficit/Hyperactivity Disorder and Autism Spectrum traits are not fixed neurological disorders but neurodevelopmental variants destabilized by the sociobiological mismatch between evolved human cognition and the pressures of modern market-based civilization. Drawing on evolutionary biology, developmental neuroscience, social epidemiology, and political economy, the paper reframes these conditions as context-contingent outcomes: traits that are biologically conserved due to their adaptive value in ancestral environments but rendered dysfunctional under chronic stress, inequality, overstimulation, environmental toxicity, and cognitive suppression endemic to industrial societies. It synthesizes evidence across prenatal programming, intergenerational stress transmission, pharmaceutical ethics, and neuroplastic adaptation to propose an ecological model in which the environment, not the brain, is the primary source of pathology. This reframing calls for systemic transformation, not individual correction, and provides a foundation for more inclusive, developmentally respectful, and ecologically coherent mental health paradigms.
注意力缺陷多动障碍(ADHD)和自闭症谱系障碍(ASD)是具有独特特征的不同神经发育状况,但它们都容易受到环境压力的影响,这些压力可能会加剧认知不匹配。本文认为,注意力缺陷/多动障碍和自闭症谱系特征并非固定的神经疾病,而是由进化而来的人类认知与现代市场经济文明压力之间的社会生物学不匹配所导致的不稳定神经发育变体。本文借鉴进化生物学、发育神经科学、社会流行病学和政治经济学,将这些状况重新界定为取决于具体情境的结果:这些特征在祖先环境中因其适应性价值而在生物学上得以保留,但在工业社会普遍存在的慢性压力、不平等、过度刺激、环境毒性和认知抑制下会变得功能失调。它综合了产前编程、代际压力传递、药物伦理和神经可塑性适应等方面的证据,提出了一种生态模型,其中环境而非大脑是病理的主要来源。这种重新界定需要系统性变革而非个体矫正,并为更具包容性、尊重发育过程且生态连贯的心理健康范式奠定了基础。