Rumiantsev Iu V, Bessmertnyĭ M Z, Vinogradov E V, Khisametdinova A I
Zh Vopr Neirokhir Im N N Burdenko. 1981 Jan-Feb(1):8-12.
The authors analyse the clinical picture and the results of nonoperative and surgical treatment of unilateral traumatic compression of the optic nerve in 23 patients with closed craniocerebral trauma. The principle role in the pathogenesis of the damage to the optic nerve is attributed to its functional compression within the optic canal as the result of impaired intratruncal circulation and edema of the nerve. This mechanism of the compression may in some cases serve as the reason for undertaking decompression of the optic nerve by resection of the superior wall of the optic canal. Eight patients were operated on, a positive ophthalmological effect was produced in 5 of them. In 15 patients who did not undergo surgery the process terminated in unilateral amaurosis.
作者分析了23例闭合性颅脑外伤患者单侧外伤性视神经受压的临床表现以及非手术和手术治疗的结果。视神经损伤发病机制中的主要作用归因于由于躯干内循环受损和神经水肿导致视神经在视神经管内的功能性受压。这种压迫机制在某些情况下可能是通过切除视神经管上壁对视神经进行减压的原因。8例患者接受了手术,其中5例产生了积极的眼科效果。15例未接受手术的患者病情以单侧黑蒙告终。