关于肠神经节缺如的发病机制。基于对实验性和临床神经节缺如的电子显微镜观察的思考。
On the pathogenesis of intestinal aganglionosis. A consideration based on electron microscopic observations of experimental and clinical aganglionoses.
作者信息
Furuta T, Sakata K, Kunieda T, Kishimoto Y
出版信息
Experientia. 1980 Sep 15;36(9):1073-4. doi: 10.1007/BF01965977.
PMID:7418843
Abstract
A close resemblance in fine structure was observed between the aganglionic colon, produced in rats by serosal application of 0.1% benzalkonium chloride solution, and the aganglionic descending colon, or more proximal segments, of Hirschsprung's disease. The pathogenesis of the disease is discussed.
摘要
通过向大鼠浆膜应用0.1%苯扎氯铵溶液产生的无神经节结肠，与先天性巨结肠病的无神经节降结肠或更靠近近端的节段之间，在精细结构上观察到了密切的相似性。本文讨论了该疾病的发病机制。
Zotero 插件