Lind O
Department of Otolaryngology-Head & Neck Surgery, Haukeland University Hospital, Bergen, Norway.
Br J Audiol. 1994 Aug-Oct;28(4-5):219-25. doi: 10.3109/03005369409086571.
Latency of the contralateral suppression of transient-evoked otoacoustic emissions (TEOAE) has been investigated using the ILO88 system for ipsilateral stimulation and recording, and a laboratory computer interface producing contralateral noise bursts. Reduction of rms amplitude in the last 10.24 ms of the response was found to produce the most consistent indication of suppression. Onset latencies varied from less than 40 ms to 140 ms, offset latency from 20 ms to more than 80 ms. Acoustic reflex latencies have not been reported with such short latencies and this makes it unlikely that this reflex can be responsible for the contralateral suppression of TEOAE. This study used contralateral stimuli at 70 dB HL. Lower level stimuli may result in increased amplitude and it is also possible that the effect of contralateral stimulation may be frequency specific. The long offset latency necessitates long intervals between stimuli with an inherent risk of variation in stimulus and recording conditions. This makes an elaborate study of the latency difficult to perform in humans.
利用ILO88系统进行同侧刺激和记录,并通过实验室计算机接口产生对侧噪声猝发,对瞬态诱发耳声发射(TEOAE)的对侧抑制潜伏期进行了研究。发现响应最后10.24毫秒内均方根振幅的降低产生了最一致的抑制指示。起始潜伏期从小于40毫秒到140毫秒不等,终止潜伏期从20毫秒到超过80毫秒不等。尚未有如此短潜伏期的听觉反射潜伏期的报道,这使得这种反射不太可能是TEOAE对侧抑制的原因。本研究使用70 dB HL的对侧刺激。较低水平的刺激可能会导致振幅增加,对侧刺激的效果也可能具有频率特异性。较长的终止潜伏期需要刺激之间有较长的间隔,存在刺激和记录条件变化的固有风险。这使得在人体中对潜伏期进行详尽研究难以开展。