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[心肌梗死急性期多水平传导障碍:一项电生理研究]

[Conduction disorders at multiple levels during the acute phase of a myocardial infarct: an electrophysiological study].

作者信息

García Burgos A, Rangel Abundis A, Castaño R, Ramos M A, Badui E

机构信息

Unidad Coronaria, Hospital de Especialidades del Centro Médico La Raza, IMSS. México, D.F.

出版信息

Arch Inst Cardiol Mex. 1993 Mar-Apr;63(2):127-32.

PMID:8503713
Abstract

Forty patients with a diagnosis of acute myocardial infarction (anterior 24, and inferior 16) were studied. Of these patients, 37.5% manifested second and third degree atrioventricular (AV) block as a complication; another 30% showed complete right bundle branch and left anterior hemiblock. Right bundle branch and left posterior hemiblock were evidenced in 12.5% of the subjects. There was 20% with complete left bundle branch block. Electrophysiologic studies were performed in all patients to assess the site of block. A direct relation was found between the surface ECG and the His bundle electrogram studies in patients with an inferior myocardial infarction and AV block, both procedures located the conduction disturbances at the AV node (suprahisian block), in contrast to patients with anteroseptal myocardial infarction whose surface ECG only showed bundle branch block or fascicular block. The His bundle electrogram registered multiple levels of AV block, 70% with troncular and infrahisian block that gave way to sudden AV block. The mechanism responsible for this block was considered to be a functional longitudinal dissociation of conduction system due to an acute ischemic injury of the His bundle, more than a sudden and simultaneous failure of all the bundle branch of His. We conclude that electrophysiologic studies are a useful procedure for identification of a group of patients with multiple AV conduction disturbances that have a less favorable prognosis than those with only suprahisian level of block.

摘要

对40例诊断为急性心肌梗死的患者(前壁24例,下壁16例)进行了研究。在这些患者中,37.5%出现二度和三度房室传导阻滞作为并发症;另外30%表现为完全性右束支传导阻滞和左前分支阻滞。12.5%的受试者出现右束支传导阻滞和左后分支阻滞。20%的患者有完全性左束支传导阻滞。对所有患者进行了电生理研究以评估阻滞部位。在下壁心肌梗死合并房室传导阻滞的患者中,发现体表心电图与希氏束电图研究之间存在直接关系,两种检查方法均将传导障碍定位在房室结(希氏束以上阻滞),而前间隔心肌梗死患者的体表心电图仅显示束支传导阻滞或分支阻滞。希氏束电图记录到多个水平的房室传导阻滞,70%为希氏束内和希氏束以下阻滞,可发展为突然的房室传导阻滞。认为这种阻滞的机制是由于希氏束急性缺血损伤导致传导系统功能性纵向分离,而非希氏束所有分支突然同时衰竭。我们得出结论,电生理研究是识别一组有多个房室传导障碍患者的有用方法,这些患者的预后比仅希氏束以上水平阻滞的患者更差。

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