臂丛神经撕脱伤后脊髓疝入相关假性脑脊膜膨出:病例报告
Spinal cord herniation into associated pseudomeningocele after brachial plexus avulsion injury: case report.
作者信息
Yokota Hiroshi, Yokoyama Kazuhiro, Noguchi Hiroshi, Uchiyama Yoshitomo
机构信息
Department of Neurosurgery, Higashiosaka City General Hospital, Higashiosaka, Japan.
出版信息
Neurosurgery. 2007 Jan;60(1):E205; discussion E205. doi: 10.1227/01.NEU.0000249195.76527.61.
OBJECTIVE
Posttraumatic spinal cord herniation is a rare condition. We describe a case of spinal cord herniation into an associated pseudomeningocele after a brachial plexus avulsion injury.
CLINICAL PRESENTATION
A 33-year-old man began to develop progressive Horner's syndrome 14 years after a brachial plexus avulsion injury. At a clinical presentation 17 years after that injury, sensory disturbance and a unilateral pyramidal sign were also evident. In addition to myelography and computed tomographic myelography findings, coronal magnetic resonance imaging scans clearly demonstrated herniation of the spinal cord into a large pseudomeningocele inside the C7-T1 intervertebral foramen. Another pseudomeningocele inside the T1-T2 intervertebral foramen was also noted.
INTERVENTION
The patient underwent a C6-T2 laminectomy, during which the spinal cord was found to be herniated through a dural defect into a pseudomeningocele at the C8 root level, and a second dural defect was also shown, with an arachnoid outpouching that included an avulsed T1 root. The spinal cord herniation was reduced and the dural defects were repaired. After surgery, the patient showed no significant neurological changes, and his condition stabilized.
CONCLUSION
Brachial plexus root avulsions may result in the formation of pseudomeningoceles and can lead to spinal cord herniation. Coronal magnetic resonance imaging is useful to demonstrate spinal cord herniation as well as pseudomeningoceles. Surgical treatment is recommended for such cases with progressive symptoms to prevent further deterioration.
目的
创伤后脊髓疝是一种罕见的病症。我们描述了一例臂丛神经撕脱伤后脊髓疝入相关假性脊膜膨出的病例。
临床表现
一名33岁男性在臂丛神经撕脱伤14年后开始出现进行性霍纳综合征。在该损伤17年后的临床表现中,感觉障碍和单侧锥体束征也很明显。除了脊髓造影和计算机断层脊髓造影结果外,冠状位磁共振成像扫描清楚地显示脊髓疝入C7 - T1椎间孔内的一个大的假性脊膜膨出。还注意到T1 - T2椎间孔内有另一个假性脊膜膨出。
干预措施
患者接受了C6 - T2椎板切除术,术中发现脊髓通过硬脑膜缺损疝入C8神经根水平的假性脊膜膨出,还发现了第二个硬脑膜缺损,伴有一个包含撕脱的T1神经根的蛛网膜袋状膨出。脊髓疝得到复位,硬脑膜缺损得到修复。术后,患者没有明显的神经功能变化,病情稳定。
结论
臂丛神经根撕脱可能导致假性脊膜膨出的形成,并可导致脊髓疝。冠状位磁共振成像有助于显示脊髓疝以及假性脊膜膨出。对于有进行性症状的此类病例,建议进行手术治疗以防止病情进一步恶化。