Takayasu K, Moriyama N, Suzuki M, Yamada T, Fukutake T, Shima Y, Kobayashi C, Musha H, Okuda K
Radiology. 1983 Apr;147(1):211-4. doi: 10.1148/radiology.147.1.6828732.
Tc-99m-phytate scanning of the liver and percutaneous transhepatic catheterization of the portal vein were performed in 33 patients--26 with cirrhosis, 3 with chronic active hepatitis, 2 with idiopathic portal hypertension, and 2 with unresolved acute hepatitis. A discrete defect in the porta hepatis area was seen in 6 of 28 patients who had portal vein pressure above 200 mm H2O. In 5 of the 6 patients with a false-positive scan, the umbilical portion of the left portal vein branch was dilated (larger than 25 x 20 mm) on the portogram, with or without a patent paraumbilical vein. The anatomical basis of this phenomenon is discussed, and it is suggested that this area be given special attention.
对33例患者进行了肝脏锝-99m植酸盐扫描及经皮经肝门静脉插管术,其中26例为肝硬化患者,3例为慢性活动性肝炎患者,2例为特发性门静脉高压患者,2例为未愈的急性肝炎患者。在门静脉压力高于200 mm H2O的28例患者中,有6例在肝门区出现离散性缺损。在6例扫描结果为假阳性的患者中,有5例在门静脉造影片上左门静脉分支的脐部扩张(大于25×20 mm),伴或不伴有副脐静脉通畅。本文讨论了这一现象的解剖学基础,并建议对该区域给予特别关注。