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腰椎穿刺后头痛综合征的磁共振成像表现:硬脑膜-脑脊膜及硬脑膜静脉窦强化异常。

MRI findings in lumbar puncture headache syndrome: abnormal dural-meningeal and dural venous sinus enhancement.

作者信息

Bakshi R, Mechtler L L, Kamran S, Gosy E, Bates V E, Kinkel P R, Kinkel W R

机构信息

University at Buffalo (SUNY) School of Medicine and Biomedical Sciences, USA.

出版信息

Clin Imaging. 1999 Mar-Apr;23(2):73-6. doi: 10.1016/s0899-7071(99)00109-6.

DOI:10.1016/s0899-7071(99)00109-6
PMID:10416079
Abstract

Intracranial hypotension (IH) is a treatable cause of persistent headaches. Persistent cerebrospinal fluid (CSF) leak at a lumbar puncture (LP) site may cause IH. We present postcontrast MRI of a patient with post-lumbar-puncture headache (LPHA) showing abnormal, intense, diffuse, symmetric, contiguous dural-meningeal (pachymeningeal) enhancement of the supratentorial and infratentorial intracranial dura, including convexities, interhemispheric fissure, tentorium, and falx. MRI also showed abnormal dural venous sinus enhancement, a new finding in LPHA, suggesting compensatory venous expansion. Thus, IH and venodilatation may play a role in the development of LPHA.

摘要

颅内低压(IH)是持续性头痛的一个可治疗病因。腰椎穿刺(LP)部位持续的脑脊液(CSF)漏可能导致颅内低压。我们展示了一位腰椎穿刺后头痛(LPHA)患者的增强后MRI,其显示幕上和幕下颅内硬脑膜出现异常、强烈、弥漫、对称且连续的硬脑膜-脑脊膜(硬脑脊膜)强化,包括脑凸面、大脑镰下间隙、小脑幕和大脑镰。MRI还显示硬脑膜静脉窦强化异常,这是LPHA中的一个新发现,提示代偿性静脉扩张。因此,颅内低压和静脉扩张可能在LPHA的发生中起作用。

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