靶向转化生长因子-β过表达：在抗Thy1肾小球肾炎中最大化血管紧张素II阻断的抗纤维化作用。 - Suppr

Suppr

超能文献

Targeting TGF-beta overexpression: maximizing the antifibrotic actions of angiotensin II blockade in anti-Thy1 glomerulonephritis.

作者信息

Peters H, Border W A, Noble N A

机构信息

Division of Nephrology, University of Utah, Salt Lake City, USA.

出版信息

Nephrol Dial Transplant. 1999;14 Suppl 4:22-3. doi: 10.1093/ndt/14.suppl_4.22.

DOI:10.1093/ndt/14.suppl_4.22

PMID:10463199

Abstract

摘要

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Targeting TGF-beta overexpression: maximizing the antifibrotic actions of angiotensin II blockade in anti-Thy1 glomerulonephritis.靶向转化生长因子-β过表达：在抗Thy1肾小球肾炎中最大化血管紧张素II阻断的抗纤维化作用。

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Angiotensin II blockade and low-protein diet produce additive therapeutic effects in experimental glomerulonephritis.血管紧张素II阻断与低蛋白饮食在实验性肾小球肾炎中产生相加治疗作用。

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Angiotensin-converting enzyme inhibition but not beta-adrenergic blockade limits transforming growth factor-beta overexpression in acute normotensive anti-thy1 glomerulonephritis.血管紧张素转换酶抑制而非β-肾上腺素能阻断可限制急性正常血压抗Thy1肾小球肾炎中转化生长因子-β的过度表达。

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Combining TGF-beta inhibition and angiotensin II blockade results in enhanced antifibrotic effect.联合抑制转化生长因子-β和阻断血管紧张素II可增强抗纤维化作用。

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Platelet inhibition limits TGF-beta overexpression and matrix expansion after induction of anti-thy1 glomerulonephritis.血小板抑制可限制抗 Thy1 肾小球肾炎诱导后的 TGF-β 过表达和基质扩张。

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NO mediates antifibrotic actions of L-arginine supplementation following induction of anti-thy1 glomerulonephritis.一氧化氮介导抗 Thy1 肾小球肾炎诱导后补充 L-精氨酸的抗纤维化作用。

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Low-dose angiotensin II receptor antagonists and angiotensin II-converting enzyme inhibitors alone or in combination for treatment of primary glomerulonephritis.低剂量血管紧张素II受体拮抗剂和血管紧张素II转换酶抑制剂单独或联合用于治疗原发性肾小球肾炎。

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Regulation of renal growth factors and clusterin by AT1 receptors during neonatal ureteral obstruction.新生儿输尿管梗阻期间AT1受体对肾脏生长因子和簇集蛋白的调节作用

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Blocking angiotensin II ameliorates proteinuria and glomerular lesions in progressive mesangioproliferative glomerulonephritis.阻断血管紧张素II可改善进行性系膜增生性肾小球肾炎的蛋白尿和肾小球病变。

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引用本文的文献

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Nephron Extra. 2012 Jan;2(1):9-16. doi: 10.1159/000335750. Epub 2012 Jan 26.

Effects of TJN-598, a new selective phosphodiesterase type IV inhibitor on anti-Thy1 nephritis in rats.一种新型选择性磷酸二酯酶 4 抑制剂 TJN-598 对大鼠抗 Thy1 肾炎的作用。

Clin Exp Nephrol. 2011 Feb;15(1):14-24. doi: 10.1007/s10157-010-0342-8. Epub 2010 Sep 4.

Extracellular matrix molecules: potential targets in pharmacotherapy.细胞外基质分子：药物治疗的潜在靶点。

Pharmacol Rev. 2009 Jun;61(2):198-223. doi: 10.1124/pr.109.001289.

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