Jarry D, Rigolet M H, Rivaud S, Bakchine S
Service d'Electrophysiologie, CHNO des Quinze Vingts, Paris.
J Fr Ophtalmol. 1999 Oct;22(8):876-80.
Cortical blindness and Balint's syndrome are two pathologies not well-known. It seems therefore interesting to report a typical patient case, suffering from Benson's posterior cortical atrophy, who presented successively both syndromes. The Balint's syndrome, which results from a bilateral parieto-occipital junction brain injury, and combines clinically a specified triad defects: a spatial disorder of attention, a psychic paralysis of gaze and an optic ataxia. The cortical blindness, which is caused by bilateral damage of the occipital lobes (Broadman area 17). Electrophysiologically, the abolition of short-latency components of visual evoked potentials and the presence of long-latency potentials are recorded. Visual strategy and visual evoked potentials are thus the only objective examinations allowing to diagnose and follow up these patient's evolution. In any case, an adequate visual rehabilitation has to be carried out in order to help the patient recovering his autonomy.
皮质盲和巴林特综合征是两种不太为人所知的病症。因此,报告一位患有本森后皮质萎缩的典型病例似乎很有意思,该患者先后出现了这两种综合征。巴林特综合征由双侧顶枕叶交界处脑损伤引起,临床上合并特定的三联征缺陷:注意力空间障碍、凝视性精神麻痹和视觉性共济失调。皮质盲是由枕叶(布罗德曼17区)双侧损伤所致。在电生理方面,可记录到视觉诱发电位短潜伏期成分消失及长潜伏期电位存在。视觉策略和视觉诱发电位因此是仅有的可用于诊断和跟踪这些患者病情进展的客观检查。无论如何,都必须进行适当的视觉康复治疗,以帮助患者恢复自主能力。