相似文献
1
Caspase inhibition and limitation of myocardial infarct size: protection against lethal reperfusion injury.
Br J Pharmacol. 2000 May;130(2):197-200. doi: 10.1038/sj.bjp.0703336.
2
Non-specific caspase inhibition reduces infarct size and improves post-ischaemic recovery in isolated ischaemic/reperfused rat hearts.
Naunyn Schmiedebergs Arch Pharmacol. 2001 Dec;364(6):501-7. doi: 10.1007/s002100100483.
3
Protective effects of caspase-9 and poly(ADP-ribose) polymerase inhibitors on ischemia-reperfusion-induced myocardial injury.
Arch Pharm Res. 2009 Jul;32(7):1037-43. doi: 10.1007/s12272-009-1709-9. Epub 2009 Jul 31.
4
Caspase inhibitor zVAD.fmk reduces infarct size after myocardial ischaemia and reperfusion in rats but not in mice.
Resuscitation. 2008 Dec;79(3):468-74. doi: 10.1016/j.resuscitation.2008.07.008. Epub 2008 Sep 20.
5
Attenuation of delayed neuronal death after mild focal ischemia in mice by inhibition of the caspase family.
J Cereb Blood Flow Metab. 1998 Mar;18(3):238-47. doi: 10.1097/00004647-199803000-00002.
6
Doxorubicin treatment activates a Z-VAD-sensitive caspase, which causes deltapsim loss, caspase-9 activity, and apoptosis in Jurkat cells.
Exp Cell Res. 2000 Jul 10;258(1):223-35. doi: 10.1006/excr.2000.4924.
7
B-type natriuretic peptide at early reperfusion limits infarct size in the rat isolated heart.
Basic Res Cardiol. 2007 Nov;102(6):529-41. doi: 10.1007/s00395-007-0672-1. Epub 2007 Sep 26.
8
Synergistic protective effect of caspase inhibitors and bFGF against brain injury induced by transient focal ischaemia.
Br J Pharmacol. 2001 Jun;133(3):345-50. doi: 10.1038/sj.bjp.0704075.
9
In vivo myocardial infarct size reduction by a caspase inhibitor administered after the onset of ischemia.
Eur J Pharmacol. 2000 Aug 18;402(1-2):139-42. doi: 10.1016/s0014-2999(00)00477-5.
10
Attenuation of increased myocardial ischaemia-reperfusion injury conferred by hypercholesterolaemia through pharmacological inhibition of the caspase-1 cascade.
Br J Pharmacol. 2003 Jan;138(2):291-300. doi: 10.1038/sj.bjp.0705098.
引用本文的文献
1
Pharmacological inhibition of apoptosis, necroptosis, and ferroptosis confers effective cardioprotection in post-myocardial infarction in rats.
Sci Rep. 2025 Aug 24;15(1):31140. doi: 10.1038/s41598-025-17275-9.
2
Therapeutic effect of mesenchymal stem cells and their derived exosomes in diseases.
Mol Biomed. 2025 Jun 4;6(1):34. doi: 10.1186/s43556-025-00277-4.
3
The Clinical Impact of Different Cryolipolysis Protocols: An Integrative Review.
Aesthetic Plast Surg. 2025 May 2. doi: 10.1007/s00266-025-04864-2.
4
Molecular Mechanisms Underlying Heart Failure and Their Therapeutic Potential.
Cells. 2025 Feb 20;14(5):324. doi: 10.3390/cells14050324.
5
Different types of cell death and their interactions in myocardial ischemia-reperfusion injury.
Cell Death Discov. 2025 Mar 5;11(1):87. doi: 10.1038/s41420-025-02372-5.
6
Metformin Alleviates Doxorubicin-Induced Cardiotoxicity via Preserving Mitochondrial Dynamics Balance and Calcium Homeostasis.
Appl Biochem Biotechnol. 2025 Apr;197(4):2713-2733. doi: 10.1007/s12010-024-05141-9. Epub 2025 Jan 10.
7
Mechanisms of postischemic cardiac death and protection following myocardial injury.
J Clin Invest. 2025 Jan 2;135(1):e184134. doi: 10.1172/JCI184134.
8
Unravelling oncosis: morphological and molecular insights into a unique cell death pathway.
Front Immunol. 2024 Aug 29;15:1450998. doi: 10.3389/fimmu.2024.1450998. eCollection 2024.
9
Targeting cell death pathways in intestinal ischemia-reperfusion injury: a comprehensive review.
Cell Death Discov. 2024 Mar 4;10(1):112. doi: 10.1038/s41420-024-01891-x.
10
High-Density Lipoproteins at the Interface between the NLRP3 Inflammasome and Myocardial Infarction.
Int J Mol Sci. 2024 Jan 20;25(2):1290. doi: 10.3390/ijms25021290.
本文引用的文献
1
Protecting the ischaemic and reperfused myocardium in acute myocardial infarction: distant dream or near reality?
Heart. 2000 Apr;83(4):381-7. doi: 10.1136/heart.83.4.381.
2
Quantitative assessment of cardiac myocyte apoptosis in tissue sections using the fluorescence-based tunel technique enhanced with counterstains.
J Immunol Methods. 1999 Aug 31;228(1-2):23-8. doi: 10.1016/s0022-1759(99)00090-3.
3
Inhibition of apoptosis induced by ischemia-reperfusion prevents inflammation.
J Clin Invest. 1999 Sep;104(5):541-9. doi: 10.1172/JCI6974.
4
Procaspase-3 and poly(ADP)ribose polymerase (PARP) are calpain substrates.
Biochem Biophys Res Commun. 1999 Sep 16;263(1):94-9. doi: 10.1006/bbrc.1999.1315.
5
Role of caspases in hypoxia-induced necrosis of rat renal proximal tubules.
J Am Soc Nephrol. 1999 Sep;10(9):1940-9. doi: 10.1681/ASN.V1091940.
6
Caspase inhibition reduces myocyte cell death induced by myocardial ischemia and reperfusion in vivo.
J Mol Cell Cardiol. 1999 Sep;31(9):1709-15. doi: 10.1006/jmcc.1999.1006.
7
Apoptosis in myocardial ischemia-reperfusion.
Ann N Y Acad Sci. 1999 Jun 30;874:412-26. doi: 10.1111/j.1749-6632.1999.tb09255.x.
8
Caspase inhibitors in prevention of apoptosis.
Herz. 1999 May;24(3):236-41. doi: 10.1007/BF03044967.
9
Mibefradil, a T-type and L-type calcium channel blocker, limits infarct size through a glibenclamide-sensitive mechanism.
Cardiovasc Drugs Ther. 1999 Apr;13(2):115-22. doi: 10.1023/a:1007732025184.
10
Calpain inhibitor-1 reduces infarct size and DNA fragmentation of myocardium in ischemic/reperfused rat heart.
J Cardiovasc Pharmacol. 1999 Apr;33(4):580-6. doi: 10.1097/00005344-199904000-00010.
Zotero 插件