适度运动可提高运动后静息状态下的温热体温调节反应阈值。
Moderate exercise increases the post exercise resting warm thermoregulatory response thresholds.
作者信息
Kenny G P, Proulx C E, Denis P M, Giesbrecht G G
机构信息
University of Ottawa, Faculty of Health Sciences, School of Human Kinetics, Ontario, Canada.
出版信息
Aviat Space Environ Med. 2000 Sep;71(9):914-9.
BACKGROUND
The purpose of this study was to evaluate the effect of exercise on the subsequent post-exercise core temperature thresholds for vasodilation and sweating.
METHODS
On two separate days, with 6 subjects (3 males and 3 females), a whole-body water-perfused suit decreased mean skin temperature until the threshold for vasoconstriction was demonstrated. Mean skin temperature was then slowly increased (approximately 5.0 degrees C x h(-1)) until thresholds for vasodilation and sweating were clearly established. Subjects were cooled by decreasing water temperature until both esophageal and mean skin temperatures returned to near baseline values. Subjects then either performed 15 min of cycle ergometry (60% V(O2max)) followed by 30 min of recovery (Exercise), or remained seated with no exercise for 45 min (Control). Subjects were then cooled again until the onset of cutaneous vasoconstriction followed by a second warming period. The core temperature thresholds for vasodilation and sweating increased significantly by 0.49 degrees C and 0.19 degrees C post-exercise, respectively (p < 0.05). In order to compare thresholds between conditions in which both esophageal and mean skin temperatures were changing, we mathematically compensated for changes in skin temperatures using the established linear cutaneous contribution of skin to the control of vasodilation and sweating (10%).
RESULTS
The calculated core temperature threshold (at a designated skin temperature of 36.0 degrees C) for vasodilation increased significantly from 36.56 +/- 0.12 degrees C to 37.11 +/- 0.21 degrees C post-exercise (p < 0.01). Likewise, the sweating threshold increased from 36.79 +/- 0.18 degrees C to 37.05 +/- 0.23 degrees C postexercise (p < 0.01). In contrast, sequential measurements, without exercise, demonstrate a time-dependent decrease (0.18 degrees C) in the sweating threshold, with no difference in the vasodilation threshold.
CONCLUSION
These data indicate that exercise has a prolonged effect by increasing the post-exercise thresholds for both warm thermoregulatory responses.
背景
本研究的目的是评估运动对运动后血管舒张和出汗的核心温度阈值的影响。
方法
在两个不同的日子里，对6名受试者（3名男性和3名女性）使用全身水灌注服降低平均皮肤温度，直到出现血管收缩阈值。然后将平均皮肤温度缓慢升高（约5.0℃×h⁻¹），直到明确建立血管舒张和出汗阈值。通过降低水温使受试者冷却，直到食管温度和平均皮肤温度均恢复到接近基线值。然后，受试者要么进行15分钟的蹬车运动（60%最大摄氧量），随后进行30分钟的恢复（运动组），要么静坐45分钟不运动（对照组）。然后再次使受试者冷却，直到皮肤血管收缩开始，接着是第二个升温期。运动后血管舒张和出汗的核心温度阈值分别显著升高了0.49℃和0.19℃（p<0.05）。为了比较食管温度和平均皮肤温度都在变化的情况下的阈值，我们使用已确定的皮肤对血管舒张和出汗控制的线性皮肤贡献（10%）对皮肤温度变化进行数学补偿。
结果
运动后血管舒张的计算核心温度阈值（在指定皮肤温度为36.0℃时）从36.56±0.12℃显著增加到37.11±0.21℃（p<0.01）。同样，出汗阈值从运动前的36.79±0.18℃增加到37.05±0.23℃（p<0.01）。相比之下，不运动的连续测量显示出汗阈值随时间下降（0.18℃），血管舒张阈值无差异。
结论
这些数据表明，运动通过提高两种温热调节反应的运动后阈值而产生持久影响。
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