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[自然分娩后子宫动脉破裂,产褥期病程异常]

[Rupture of the uterine artery after spontaneous delivery with unusual course in childbed].

作者信息

Becker R, Kowalsky B L, Hatzmann W

机构信息

Abteilung für Gynäkologie und Geburtshilfe. St.-Bernhard Hospital Kamp-Lintfort, Germany.

出版信息

Z Geburtshilfe Neonatol. 2002 May-Jun;206(3):107-13. doi: 10.1055/s-2002-32643.

Abstract

Haemorrhage from ruptured uterine vessels is a rare but life-threatening complication during pregnancy. The high rate of mortality associated with this condition is correlated to the rapidity of haematoma formation. The dynamics of this event become particularly evident at the time of birth when the uterus requires one-fifth of the cardiac output. This high rate of flow emphasizes the possible fatal consequences of delayed therapeutical intervention. Information available on the aetiology and pathogenesis of such events is poor, especially if an involvement of cervical vessels or extraperitoneal (incomplete) rupture of the uterus can be excluded. Diagnosis and therapy are based on the classical clinical symptoms of acute abdominal pain-and-shock symptomatology. We report on a 31-year old patient after spontaneous delivery and initially uneventful puerperium. She had to be laparotomised on the third day post partum because of pain and haemoglobin deficiency. We found a large retroperitoneal (supralevatorial) haematoma caused by ruptured lateral branches of the left arteria uterina. The uterus was preserved after haematoma removal and revision and suture of the bleeding vessels. The case reported on shows that peripartal rupture of arterial uterine vessels may turn out to be clinically relevant only after a relatively long time (> 72 hours). Only four comparable cases have been reported in the literature, the course being different from that in all the other cases. In all the documented cases arterial uterine haemorrhages resulted in short-term acute aggravation of the symptomatology with severe abdominal pain, haemodynamic collapse and simultaneous haemoglobin deficit. The case under report in the present paper was at first clinically occult and eventually developed a pathology that had not been described before. Its specific significance lies in illustrating the need for re-evaluating such initially hidden cases while paying special attention to ruptured uterine vessels.

摘要

妊娠期间子宫血管破裂出血是一种罕见但危及生命的并发症。与这种情况相关的高死亡率与血肿形成的速度有关。当子宫需要五分之一的心输出量时,这种情况在分娩时变得尤为明显。这种高流量强调了延迟治疗干预可能产生的致命后果。关于此类事件的病因和发病机制的可用信息很少,特别是如果可以排除宫颈血管受累或子宫腹膜外(不完全)破裂的情况。诊断和治疗基于急性腹痛和休克症状的经典临床症状。我们报告了一名31岁的患者,她自然分娩后最初产褥期正常。产后第三天,由于疼痛和血红蛋白缺乏,她不得不接受剖腹手术。我们发现一个由左子宫动脉侧支破裂引起的巨大腹膜后(阔韧带上方)血肿。血肿清除以及对出血血管进行修复和缝合后,保留了子宫。所报告的病例表明,子宫动脉血管的围产期破裂可能在相对较长时间(>72小时)后才在临床上显现出相关性。文献中仅报道了4例类似病例,其病程与所有其他病例不同。在所有已记录的病例中,子宫动脉出血导致症状在短期内急性加重,伴有严重腹痛、血流动力学崩溃和同时出现的血红蛋白缺乏。本文所报告的病例起初在临床上隐匿,最终发展出一种以前未描述过的病理情况。其特殊意义在于说明需要重新评估此类最初隐匿的病例,同时特别关注子宫血管破裂情况。

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