Kretz A, Da Rous H O, Palumbo J R
Br Heart J. 1975 Feb;37(2):136-49. doi: 10.1136/hrt.37.2.136.
The underlying mechanism of bradycardia-dependent bundle-branch and paroxysmal atrioventricular block appears to be enhancement of phase-4 depolarization in a branch or in a natural or acquired monofascicular pathway. Clinical records of these forms of impaired conduction occurring in the bundle-branches, with either longer or shorter cardiac cycle lengths, are presented and analysed. These also include the combination of Mobitz typw I atrioventricular block with variable degrees of bundle-branch block, as a representative example of narrow ventricular escape beats firing in the zone where prominent diastolic depolarization is present.
心动过缓依赖性束支传导阻滞和阵发性房室传导阻滞的潜在机制似乎是分支或天然或后天单束支传导途径中4相去极化增强。本文展示并分析了束支出现的这些传导障碍形式在心动周期长短不同时的临床记录。这些记录还包括莫氏I型房室传导阻滞合并不同程度束支传导阻滞的情况，以此作为在存在显著舒张期去极化区域出现窄QRS波逸搏心律的一个典型例子。
