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AP-1 as a regulator of cell life and death.
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Reaching a genetic and molecular understanding of skeletal development.
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RANKL maintains bone homeostasis through c-Fos-dependent induction of interferon-beta.
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Impaired postnatal hepatocyte proliferation and liver regeneration in mice lacking c-jun in the liver.
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Genetic control of skeletal development.
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AP-1 in mouse development and tumorigenesis.
Oncogene. 2001 Apr 30;20(19):2401-12. doi: 10.1038/sj.onc.1204389.
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Fra-1 replaces c-Fos-dependent functions in mice.
Genes Dev. 2000 Nov 1;14(21):2695-700. doi: 10.1101/gad.187900.
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Placental vascularisation requires the AP-1 component fra1.
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Overexpression of DeltaFosB transcription factor(s) increases bone formation and inhibits adipogenesis.
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Fosl1 is a transcriptional target of c-Fos during osteoclast differentiation.
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