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分子层面的发现改变了我们对炎症性肠病的看法。一篇从科学、临床和实验室角度进行的综述。

Molecular discoveries alter our view of inflammatory bowel disease. A review from scientific, clinical, and laboratory perspectives.

作者信息

Staros Eric B

机构信息

Quest Diagnostics, Department of Pathology, Rush-Presbyterian Medical School, Chicago, IL, USA.

出版信息

Am J Clin Pathol. 2003 Apr;119(4):524-39. doi: 10.1309/7W3E-TL9N-Q9RH-HX61.

Abstract

Within the past decade, knowledge of the molecular basis of inflammatory bowel disease (IBD), including Crohn disease (CD) and ulcerative colitis, has advanced owing to the explosive growth of research involving the human genome. Furthermore, a shared interest between molecular biologists and clinical researchers has contributed to an emerging understanding of IBD. Nucleotide-binding oligomerization domain 2 (NOD2) belongs to an apoptotic regulatory family of genes and has been linked to CD. In addition, research into nuclear factor kappa B (NF-kappa B), the proteasome, interleukins, and tumor necrosis factor alpha has improved our understanding of IBD. Our understanding of these molecules and other scientific discoveries offers hope for new diagnostic tests and therapeutic agents. In the future, genetic markers will predict disease susceptibility, therapeutic responsiveness, and long-term sequelae of modern therapeutics. Also on the horizon, molecular markers promise to define disease heterogeneity, thereby providing a rational basis for patient-specific therapies. The molecular discoveries that are changing our views of IBD will affect the clinician, the laboratory professional, and the patient.

摘要

在过去十年中,由于涉及人类基因组的研究呈爆发式增长,对包括克罗恩病(CD)和溃疡性结肠炎在内的炎症性肠病(IBD)分子基础的认识取得了进展。此外,分子生物学家和临床研究人员之间的共同兴趣促成了对IBD的新认识。核苷酸结合寡聚化结构域2(NOD2)属于凋亡调节基因家族,与CD有关。此外,对核因子κB(NF-κB)、蛋白酶体、白细胞介素和肿瘤坏死因子α的研究增进了我们对IBD的理解。我们对这些分子以及其他科学发现的理解为新的诊断测试和治疗药物带来了希望。未来,基因标记将预测疾病易感性、治疗反应性以及现代治疗方法的长期后遗症。同样即将出现的是,分子标记有望界定疾病的异质性,从而为针对患者的治疗提供合理依据。正在改变我们对IBD看法的分子发现将影响临床医生、实验室专业人员和患者。

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