Norris E S, Woodruff R C
Department of Biological Sciences, Bowling Green State University, OH 43403.
Mutat Res. 1992 Sep;269(1):63-72. doi: 10.1016/0027-5107(92)90161-t.
This study supplied no evidence that P-M hybrid dysgenesis is a general release mechanisms for transposon movement. Newly induced mutations (23 singed, three yellow, and one white) were generated by hybrid dysgenesis and were molecularly analyzed for the presence or absence of P element insertions. Only one dysgenically-induced insertion mutation out of 27 analyzed was the result of a non-P insert; this frequency is not statistically above the non-dysgenic control level. Thus, it appears that individual transposable elements families are independently regulated.
本研究没有提供证据表明P-M杂种不育是转座子移动的一种普遍释放机制。通过杂种不育产生了新诱导的突变(23个焦刚毛、3个黄体和1个白眼),并对其进行分子分析以检测P因子插入的有无。在分析的27个突变中,只有1个由杂种不育诱导的插入突变是非P插入的结果;这个频率在统计学上并不高于非杂种不育对照水平。因此,似乎各个转座子家族是独立调控的。