[甲状旁腺功能不全时注射甲状旁腺素后甲状旁腺激素对神经分泌细胞中45Ca2+蓄积及血液中血管加压素含量的影响]
[Effects of parathormone on 45Ca2+ accumulation in neurosecretory cells and contents of vasopressin in blood after administration of parathyroidin in parathyroid gland insufficiency].
作者信息
Khudaverdian D N, Asratian A A
出版信息
Biull Eksp Biol Med. 1992 Mar;113(3):230-2.
The functional state of hypothalamic-hypophyseal-neurosecretory complex was studied in parathyroidin injection and hypofunction of the parathyroid glands. In these conditions vasopressin levels in blood of the rats increased. The incubation of the hypothalamus with parathormone and 45Ca2+, as well as the injection of parathyroidin increased 45Ca2+ transfer to the neurosecretory cells and vasopressin release in the blood. Various mechanisms of similar influence of the parathormone and hypofunction of the parathyroid glands on a vasopressin level in blood are discussed.
在甲状旁腺素注射及甲状旁腺功能减退的情况下，对下丘脑 - 垂体 - 神经分泌复合体的功能状态进行了研究。在这些条件下，大鼠血液中的血管加压素水平升高。用甲状旁腺激素和45Ca2 +孵育下丘脑，以及注射甲状旁腺素，均可增加45Ca2 +向神经分泌细胞的转运及血液中血管加压素的释放。文中讨论了甲状旁腺激素和甲状旁腺功能减退对血液中血管加压素水平产生类似影响的各种机制。
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