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Cardiovascular responses to hypoxic stimulation of the carotid bodies.

作者信息

DOWNING S E, REMENSNYDER J P, MITCHELL J H

出版信息

Circ Res. 1962 Apr;10:676-85. doi: 10.1161/01.res.10.4.676.

DOI:10.1161/01.res.10.4.676

PMID:13887639

Abstract

摘要

相似文献

Cardiovascular responses to hypoxic stimulation of the carotid bodies.

Circ Res. 1962 Apr;10:676-85. doi: 10.1161/01.res.10.4.676.

Comparison of the reflex responses elicited by stimulation of the separately perfused carotid and aortic body chemoreceptors in the dog.

J Physiol. 1966 Jan;182(2):379-403. doi: 10.1113/jphysiol.1966.sp007828.

Interplay among carotid sinus, cardiopulmonary, and carotid body reflexes in dogs.

Am J Physiol. 1976 Jan;230(1):19-24. doi: 10.1152/ajplegacy.1976.230.1.19.

Responses of the vasomotor center to hypoxia after denervation of carotid and aortic bodies.

Am J Physiol. 1951 Jul;166(1):45-54. doi: 10.1152/ajplegacy.1951.166.1.45.

[Orthosympathetic response of sinocarotid glomus origin after local and general acetylcholine injections in dogs].

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Role of carotid and aortic bodies in mediating the increase in cardiac output during anoxemia.

Am J Physiol. 1962 Jul;203:133-6. doi: 10.1152/ajplegacy.1962.203.1.133.

Influence of the carotid chemoceptor reflexes on the heart rate in systemic anoxia.

Arch Int Pharmacodyn Ther. 1956 Mar 1;105(3-4):477-88.

Effect of cholinergic antagonists on sympathetic ganglionic transmission of vasomotor reflexes from the carotid baroreceptors and chemoreceptors of the dog.

J Physiol. 1978 Apr;277:379-85. doi: 10.1113/jphysiol.1978.sp012278.

Observations on carotid body chemoreceptor activity and cervical sympathetic discharge in the cat.

J Physiol. 1967 Jun;190(3):413-24. doi: 10.1113/jphysiol.1967.sp008218.

Cardiovascular alterations by chronic intermittent hypoxia: importance of carotid body chemoreflexes.

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MECHANISMS FOR CARDIAC STIMULATION DURING ANOXEMIA IN THE MODIFIED HEART-LUNG PREPARATIONS.

Pflugers Arch Gesamte Physiol Menschen Tiere. 1965;282:209-24. doi: 10.1007/BF00412709.

THE EFFECTS OF ARTIFICIALLY INDUCED HYPERVENTILATION ON THE PRIMARY CARDIAC REFLEX RESPONSE TO STIMULATION OF THE CAROTID BODIES IN THE DOG.

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Cardiovascular responses to hypoxic stimulation of the carotid bodies.

作者信息

DOWNING S E, REMENSNYDER J P, MITCHELL J H

出版信息

Circ Res. 1962 Apr;10:676-85. doi: 10.1161/01.res.10.4.676.

DOI:10.1161/01.res.10.4.676

PMID:13887639

Abstract

摘要

相似文献

Cardiovascular responses to hypoxic stimulation of the carotid bodies.

Circ Res. 1962 Apr;10:676-85. doi: 10.1161/01.res.10.4.676.

Comparison of the reflex responses elicited by stimulation of the separately perfused carotid and aortic body chemoreceptors in the dog.

J Physiol. 1966 Jan;182(2):379-403. doi: 10.1113/jphysiol.1966.sp007828.

Interplay among carotid sinus, cardiopulmonary, and carotid body reflexes in dogs.

Am J Physiol. 1976 Jan;230(1):19-24. doi: 10.1152/ajplegacy.1976.230.1.19.

Responses of the vasomotor center to hypoxia after denervation of carotid and aortic bodies.

Am J Physiol. 1951 Jul;166(1):45-54. doi: 10.1152/ajplegacy.1951.166.1.45.

[Orthosympathetic response of sinocarotid glomus origin after local and general acetylcholine injections in dogs].

C R Seances Soc Biol Fil. 1971;165(9):1826-31.

Role of carotid and aortic bodies in mediating the increase in cardiac output during anoxemia.

Am J Physiol. 1962 Jul;203:133-6. doi: 10.1152/ajplegacy.1962.203.1.133.

Influence of the carotid chemoceptor reflexes on the heart rate in systemic anoxia.

Arch Int Pharmacodyn Ther. 1956 Mar 1;105(3-4):477-88.

Effect of cholinergic antagonists on sympathetic ganglionic transmission of vasomotor reflexes from the carotid baroreceptors and chemoreceptors of the dog.

J Physiol. 1978 Apr;277:379-85. doi: 10.1113/jphysiol.1978.sp012278.

Observations on carotid body chemoreceptor activity and cervical sympathetic discharge in the cat.

J Physiol. 1967 Jun;190(3):413-24. doi: 10.1113/jphysiol.1967.sp008218.

Cardiovascular alterations by chronic intermittent hypoxia: importance of carotid body chemoreflexes.

Clin Exp Pharmacol Physiol. 2005 May-Jun;32(5-6):447-9. doi: 10.1111/j.1440-1681.2005.04209.x.

引用本文的文献

Change in sleep, gastrointestinal symptoms, and mood states at high altitude (4500m) for 6 months.

Sleep Breath. 2025 Jan 13;29(1):72. doi: 10.1007/s11325-024-03237-4.

Tibetans exhibit lower hemoglobin concentration and decreased heart response to hypoxia during poikilocapnia at intermediate altitude relative to Han Chinese.

Front Physiol. 2024 May 9;15:1334874. doi: 10.3389/fphys.2024.1334874. eCollection 2024.

The carotid chemoreceptor contributes to the elevated arterial stiffness and vasoconstrictor outflow in chronic obstructive pulmonary disease.

J Physiol. 2018 Aug;596(15):3233-3244. doi: 10.1113/JP275762. Epub 2018 Apr 11.

Hypovolemia explains the reduced stroke volume at altitude.

Physiol Rep. 2013 Oct;1(5):e00094. doi: 10.1002/phy2.94. Epub 2013 Oct 2.

Hypoxia stress test reveals exaggerated cardiovascular effects in hypertensive rats after exposure to the air pollutant acrolein.

Toxicol Sci. 2013 Apr;132(2):467-77. doi: 10.1093/toxsci/kft008. Epub 2013 Jan 18.

A key circulatory defence against asphyxia in infancy--the heart of the matter!

J Physiol. 2012 Dec 1;590(23):6157-65. doi: 10.1113/jphysiol.2012.239145. Epub 2012 Sep 24.

Serotonin nerve terminals in the dorsomedial medulla facilitate sympathetic and ventilatory responses to hemorrhage and peripheral chemoreflex activation.

Am J Physiol Regul Integr Comp Physiol. 2011 Nov;301(5):R1367-79. doi: 10.1152/ajpregu.00576.2010. Epub 2011 Aug 3.

The effects of acute hypoxia on the systemic venous and arterial systems and on myocardial contractile force.

J Clin Invest. 1962 Jul;41(7):1553-63. doi: 10.1172/JCI104612.

MECHANISMS FOR CARDIAC STIMULATION DURING ANOXEMIA IN THE MODIFIED HEART-LUNG PREPARATIONS.

Pflugers Arch Gesamte Physiol Menschen Tiere. 1965;282:209-24. doi: 10.1007/BF00412709.

THE EFFECTS OF ARTIFICIALLY INDUCED HYPERVENTILATION ON THE PRIMARY CARDIAC REFLEX RESPONSE TO STIMULATION OF THE CAROTID BODIES IN THE DOG.

J Physiol. 1963 Oct;168(4):872-89. doi: 10.1113/jphysiol.1963.sp007228.