Location, location, location: regulation of breast cancer progression by the microenvironment.
作者信息
Lee Adrian V
机构信息
Breast Center, Baylor College of Medicine and the Methodist Hospital, Houston, Texas, USA.
出版信息
Breast Cancer Res. 2004;6(6):279-80. doi: 10.1186/bcr957. Epub 2004 Oct 8.
Abstract
摘要
相似文献
1
Location, location, location: regulation of breast cancer progression by the microenvironment.位置，位置，还是位置：微环境对乳腺癌进展的调控
Breast Cancer Res. 2004;6(6):279-80. doi: 10.1186/bcr957. Epub 2004 Oct 8.
2
The tumor suppressor Smad4 is required for transforming growth factor beta-induced epithelial to mesenchymal transition and bone metastasis of breast cancer cells.肿瘤抑制因子Smad4是转化生长因子β诱导乳腺癌细胞上皮-间质转化和骨转移所必需的。
Cancer Res. 2006 Feb 15;66(4):2202-9. doi: 10.1158/0008-5472.CAN-05-3560.
3
Contributions of mouse biology to breast cancer research.小鼠生物学对乳腺癌研究的贡献。
Comp Med. 2002 Feb;52(1):12-31.
4
[Stem cells and epithelial cancers: the example of breast cancer].[干细胞与上皮性癌：以乳腺癌为例]
Ann Pathol. 2008 Nov;28 Spec No 1(1):S30-2. doi: 10.1016/j.annpat.2008.09.005. Epub 2008 Oct 17.
5
RbAp46 inhibits estrogen-stimulated progression of neoplastigenic breast epithelial cells.RbAp46抑制雌激素刺激的致瘤性乳腺上皮细胞的进展。
Anticancer Res. 2007 Sep-Oct;27(5A):3205-9.
6
Breast cancer cell-derived fibroblast growth factor 2 and vascular endothelial growth factor are chemoattractants for bone marrow stromal stem cells.乳腺癌细胞衍生的成纤维细胞生长因子2和血管内皮生长因子是骨髓基质干细胞的趋化因子。
Ann Surg. 2008 Feb;247(2):310-4. doi: 10.1097/SLA.0b013e31816401d5.
7
Caveolin-1 down-regulation activates estrogen receptor alpha expression and leads to 17beta-estradiol-stimulated mammary tumorigenesis.小窝蛋白-1的下调激活雌激素受体α的表达，并导致17β-雌二醇刺激的乳腺肿瘤发生。
Anticancer Res. 2005 Jan-Feb;25(1A):369-75.
8
Metastasis tumor antigen family proteins during breast cancer progression and metastasis in a reliable mouse model for human breast cancer.在一种可靠的人类乳腺癌小鼠模型中，转移肿瘤抗原家族蛋白在乳腺癌进展和转移过程中的作用
Clin Cancer Res. 2006 Mar 1;12(5):1479-86. doi: 10.1158/1078-0432.CCR-05-1519.
9
Transforming growth factor-beta regulates mammary carcinoma cell survival and interaction with the adjacent microenvironment.转化生长因子-β调节乳腺癌细胞的存活以及与邻近微环境的相互作用。
Cancer Res. 2008 Mar 15;68(6):1809-19. doi: 10.1158/0008-5472.CAN-07-5597.
10
Tumor microenvironment: the role of the tumor stroma in cancer.肿瘤微环境：肿瘤基质在癌症中的作用
J Cell Biochem. 2007 Jul 1;101(4):805-15. doi: 10.1002/jcb.21159.
引用本文的文献
1
Identification of the metabolic protein ATP5MF as a potential therapeutic target of TNBC.鉴定代谢蛋白 ATP5MF 为三阴性乳腺癌的潜在治疗靶点。
J Transl Med. 2024 Oct 14;22(1):932. doi: 10.1186/s12967-024-05692-9.
本文引用的文献
1
Targeted disruption of beta1-integrin in a transgenic mouse model of human breast cancer reveals an essential role in mammary tumor induction.在人类乳腺癌转基因小鼠模型中对β1整合素进行靶向破坏揭示了其在乳腺肿瘤诱导中的关键作用。
Cancer Cell. 2004 Aug;6(2):159-70. doi: 10.1016/j.ccr.2004.06.025.
2
Molecular characterization of the tumor microenvironment in breast cancer.乳腺癌肿瘤微环境的分子特征
Cancer Cell. 2004 Jul;6(1):17-32. doi: 10.1016/j.ccr.2004.06.010.
3
Inhibition of breast cancer metastasis by selective synthetic polypeptide against CXCR4.选择性合成多肽对CXCR4的抑制作用与乳腺癌转移
Cancer Res. 2004 Jun 15;64(12):4302-8. doi: 10.1158/0008-5472.CAN-03-3958.
4
The stroma as a crucial target in rat mammary gland carcinogenesis.基质作为大鼠乳腺癌发生过程中的关键靶点。
J Cell Sci. 2004 Mar 15;117(Pt 8):1495-502. doi: 10.1242/jcs.01000. Epub 2004 Mar 2.
5
Tumor reversion: correction of malignant behavior by microenvironmental cues.肿瘤逆转：通过微环境线索纠正恶性行为。
Int J Cancer. 2003 Dec 10;107(5):688-95. doi: 10.1002/ijc.11491.
6
Myoepithelial mRNA expression profiling reveals a common tumor-suppressor phenotype.肌上皮细胞mRNA表达谱揭示了一种常见的肿瘤抑制表型。
Exp Mol Pathol. 2003 Apr;74(2):113-22. doi: 10.1016/s0014-4800(03)00011-x.
Zotero 插件