Bergeron P, Coulon P, De Chaumaray T, Ruiz M, Mariotti F, Gay J, Mangialardi N, Costa P, Serreo E, Cavazzini C, Tuccimei I
Department of Thoracic and Cardiovascular Surgery Saint Joseph Hospital, Marseille, France.
J Cardiovasc Surg (Torino). 2005 Apr;46(2):141-7.
We describe our experience in endovascular repair of Thoracic Aortic Aneurysms and Dissections (TAAD) involving the aortic arch in high risk patients (HRP).
Twenty-nine patients presented with TAAD involving the aortic arch and were treated by endovascular exclusion. Pathologies were as follows: atherosclerotic aneurysms of the descending thoracic aorta in 15 cases, acute Stanford type A dissections in 6 cases, Stanford type B dissections in 7 cases (1 acute), and 1 false aneurysm of the ascending aorta. Total-arch transpositions of all supra-aortic vessels (aortic debranching) to the ascending aorta were done in 11 cases throught median sternotomy. We performed carotido-carotid bypass (hemi-arch transposition) in 16 patients by cervicotomy. Secondary to surgical transpositions, we placed endovascular stentgrafts in all but 2 patients for final exclusion, the 2 remaining being planned for later exclusion. The Talent, Excluder, TAG and Zenith endografts were used in 12, 3, 1 and 4 cases respectively. Banding technique was associated in some cases.
All surgical transpositions were successful although 1 led to a minor stroke (1/29=3.5%), which worsened to major stroke after endovascular exclusion. Endovascular procedures were performed in all but one case (26/27=96.3%). Two patients (2/26=7.7%) died from catheterization related complications after endovascular exclusion (iliac rupture and left ventricle perforation). One patient had a delayed minor stroke (1/26=3.8%). Recirculation was found in 13.3% (2/15) of aneurysms and 27.3% of thoracic false channels. During a mean follow-up of 15.7 months (13 days to 45.5 months), 1 patient (1/26=3.8%) who had preoperative chronic pulmonary failure died at 6 months from respiratory worsening. We observed one case (3.8%) of unilateral limb palsy unrelated to cerebral ischemia, which we successfully treated by cerebrospinal fluid (CSF) drainage. No stent-related complication was seen. One new type 1 endoleak appeared at 12 months on an aneurysm, which resolved after stentgraft extension. Three thoracic dissection false channels remained patent during follow-up, of which one was retrograde originating distally in the descending aorta.
Secondary endovascular exclusion of thoracic aortic diseases involving the arch in HRP is made feasible thanks to the preliminary aortic debranching. Total-arch transposition may be of greater interest in case of proximal neck length uncertainty and potential embolization from the aortic arch. Mid-term results are good although patients must be followed carefully to detect aortic recirculation and enlargement.
我们描述了在高危患者(HRP)中对累及主动脉弓的胸主动脉瘤和夹层(TAAD)进行血管腔内修复的经验。
29例患者患有累及主动脉弓的TAAD,并接受了血管腔内隔绝术治疗。病变情况如下:降主动脉粥样硬化性动脉瘤15例,急性Stanford A型夹层6例,Stanford B型夹层7例(1例急性),升主动脉假性动脉瘤1例。11例患者通过正中胸骨切开术将所有主动脉弓上血管进行全弓转位(主动脉去分支)至升主动脉。我们通过颈部切开术对16例患者进行了颈动脉 - 颈动脉旁路移植术(半弓转位)。在手术转位后,除2例患者计划后期进行隔绝外,其余所有患者均置入血管腔内支架移植物进行最终隔绝。分别有12例、3例、1例和4例使用了Talent、Excluder、TAG和Zenith血管腔内移植物。部分病例采用了束带技术。
所有手术转位均成功,尽管1例导致轻微卒中(1/29 = 3.5%),在血管腔内隔绝后恶化成严重卒中。除1例患者外,其余所有患者均进行了血管腔内操作(26/27 = 96.3%)。2例患者(2/26 = 7.7%)在血管腔内隔绝后死于与导管插入相关的并发症(髂动脉破裂和左心室穿孔)。1例患者发生延迟性轻微卒中(1/26 = 3.8%)。在13.3%(2/15)的动脉瘤和27.3%的胸段假腔中发现了再循环。在平均15.7个月(13天至45.5个月)的随访期间内,1例术前患有慢性肺功能衰竭的患者在6个月时因呼吸功能恶化死亡。我们观察到1例(3.8%)与脑缺血无关 的单侧肢体麻痹,通过脑脊液(CSF)引流成功治疗。未观察到与支架相关的并发症。1例动脉瘤在12个月时出现1例新的I型内漏,在延长支架移植物后消失。3个胸段夹层假腔在随访期间保持通畅,其中1个为逆行性,起源于降主动脉远端。
由于初步的主动脉去分支,使得在高危患者中对累及主动脉弓的胸主动脉疾病进行二期血管腔内隔绝成为可能。在近端颈部长度不确定和存在来自主动脉弓潜在栓塞的情况下,全弓转位可能更具优势。尽管必须对患者进行仔细随访以检测主动脉再循环和扩大情况,但中期结果良好。