相似文献
1
FGF signaling in the developing endochondral skeleton.
Cytokine Growth Factor Rev. 2005 Apr;16(2):205-13. doi: 10.1016/j.cytogfr.2005.02.003. Epub 2005 Apr 1.
2
Recent research on the growth plate: Advances in fibroblast growth factor signaling in growth plate development and disorders.
J Mol Endocrinol. 2014 Aug;53(1):T11-34. doi: 10.1530/JME-14-0012.
3
FGF signaling in skeletal development.
Front Biosci. 1998 Aug 1;3:d781-94. doi: 10.2741/a321.
4
Fibroblast growth factors in skeletal development.
Curr Top Dev Biol. 2019;133:195-234. doi: 10.1016/bs.ctdb.2018.11.020. Epub 2019 Jan 3.
5
FGFs in endochondral skeletal development.
J Cell Biochem. 2010 Aug 1;110(5):1046-57. doi: 10.1002/jcb.22629.
6
Roles of FGF signaling in skeletal development and human genetic diseases.
Front Biosci. 2005 May 1;10:1961-76. doi: 10.2741/1671.
7
Signaling by fibroblast growth factors (FGF) and fibroblast growth factor receptor 2 (FGFR2)-activating mutations blocks mineralization and induces apoptosis in osteoblasts.
J Cell Biol. 2000 Jun 12;149(6):1297-308. doi: 10.1083/jcb.149.6.1297.
8
Role of FGFs/FGFRs in skeletal development and bone regeneration.
J Cell Physiol. 2012 Dec;227(12):3731-43. doi: 10.1002/jcp.24083.
9
FGF signaling pathways in endochondral and intramembranous bone development and human genetic disease.
Genes Dev. 2002 Jun 15;16(12):1446-65. doi: 10.1101/gad.990702.
10
Fibroblast growth factor signaling controlling bone formation: an update.
Gene. 2012 Apr 25;498(1):1-4. doi: 10.1016/j.gene.2012.01.086. Epub 2012 Feb 9.
引用本文的文献
1
PTPN11 in cartilage development, adult homeostasis, and diseases.
Bone Res. 2025 May 16;13(1):53. doi: 10.1038/s41413-025-00425-0.
2
Advances in the mechanism and therapies of achondroplasia.
Genes Dis. 2024 Sep 24;12(4):101436. doi: 10.1016/j.gendis.2024.101436. eCollection 2025 Jul.
3
Posttranslational Modification in Bone Homeostasis and Osteoporosis.
MedComm (2020). 2025 Apr 1;6(4):e70159. doi: 10.1002/mco2.70159. eCollection 2025 Apr.
4
Achondroplasia current concept of orthopaedic management.
J Child Orthop. 2024 Aug 27;18(5):461-476. doi: 10.1177/18632521241269340. eCollection 2024 Oct.
5
Cell signaling and transcriptional regulation of osteoblast lineage commitment, differentiation, bone formation, and homeostasis.
Cell Discov. 2024 Jul 2;10(1):71. doi: 10.1038/s41421-024-00689-6.
6
Chronic Iodine Intake Excess Damages the Structure of Articular Cartilage and Epiphyseal Growth Plate.
Biol Trace Elem Res. 2024 Sep;202(9):4078-4086. doi: 10.1007/s12011-023-03985-6. Epub 2023 Dec 7.
8
Achondroplasia: Clinical, Radiological and Molecular Profile from Rare Disease Centre, India.
J Pediatr Genet. 2021 Jul 7;12(1):42-47. doi: 10.1055/s-0041-1731684. eCollection 2023 Mar.
9
Cranial Base Synchondrosis: Chondrocytes at the Hub.
Int J Mol Sci. 2022 Jul 15;23(14):7817. doi: 10.3390/ijms23147817.
10
Bioengineering Approaches for Delivering Growth Factors: A Focus on Bone and Cartilage Regeneration.
Bioengineering (Basel). 2022 May 20;9(5):223. doi: 10.3390/bioengineering9050223.
本文引用的文献
1
Mutations that cause osteoglophonic dysplasia define novel roles for FGFR1 in bone elongation.
Am J Hum Genet. 2005 Feb;76(2):361-7. doi: 10.1086/427956. Epub 2004 Dec 28.
2
A further mutation of the FGFR2 tyrosine kinase domain in mild Crouzon syndrome.
Eur J Hum Genet. 2005 Apr;13(4):503-5. doi: 10.1038/sj.ejhg.5201325.
3
Biochemical analysis of pathogenic ligand-dependent FGFR2 mutations suggests distinct pathophysiological mechanisms for craniofacial and limb abnormalities.
Hum Mol Genet. 2004 Oct 1;13(19):2313-24. doi: 10.1093/hmg/ddh235. Epub 2004 Jul 28.
4
Stat1 controls postnatal bone formation by regulating fibroblast growth factor signaling in osteoblasts.
J Biol Chem. 2004 Jun 25;279(26):27743-52. doi: 10.1074/jbc.M314323200. Epub 2004 Apr 8.
5
Crouzonodermoskeletal syndrome.
J Clin Pediatr Dent. 2004 Winter;28(2):173-6. doi: 10.17796/jcpd.28.2.72m01l5g50448548.
6
Muenke syndrome.
Childs Nerv Syst. 2004 May;20(5):297-301. doi: 10.1007/s00381-003-0906-y. Epub 2004 Feb 10.
7
Constitutive activation of MEK1 in chondrocytes causes Stat1-independent achondroplasia-like dwarfism and rescues the Fgfr3-deficient mouse phenotype.
Genes Dev. 2004 Feb 1;18(3):290-305. doi: 10.1101/gad.1179104.
8
Overexpression of FGFR3, Stat1, Stat5 and p21Cip1 correlates with phenotypic severity and defective chondrocyte differentiation in FGFR3-related chondrodysplasias.
Bone. 2004 Jan;34(1):26-36. doi: 10.1016/j.bone.2003.09.002.
9
Defective bone mineralization and osteopenia in young adult FGFR3-/- mice.
Hum Mol Genet. 2004 Feb 1;13(3):271-84. doi: 10.1093/hmg/ddh034. Epub 2003 Dec 17.
10
Molecular basis for the treatment of achondroplasia.
Horm Res. 2003;60 Suppl 3:60-4. doi: 10.1159/000074503.
Zotero 插件