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关于前庭器官感知声音刺激的能力。

On the capability of the vestibular apparatus to perceive sound stimuli.

作者信息

Ribarić K, Kekić B, Dergenc R

机构信息

Department of Otorhinolaryngology and Head and Neck Surgery, Zagreb, Yugoslavia.

出版信息

Acta Otolaryngol. 1992;112(2):221-4. doi: 10.1080/00016489.1992.11665408.

DOI:10.1080/00016489.1992.11665408
PMID:1604983
Abstract

This paper discusses the capacity of sound perception in 5 profoundly deaf subjects in whom a "fenestra" was previously formated on the lateral semicircular canal. The ability of sound and vibration perception after fenestra formation in profoundly deaf subjects with normal function of the vestibular apparatus has been described previously. The experiments for this procedure were done on pigeons by Wit & Bleeker. The subjects aged from 22 to 34 years. Causes of deafness varied. Their hearing threshold was bilaterally higher than 95 dB HL and their vestibular apparatus was of normal excitability bilaterally when tested with a caloric test. In all 5 patients the ear conduction hearing threshold remained the same, but the ability of vibration perception improved significantly, and ranged postoperatively between 30 and 45 dB HL. The frequency dynamic range was from 125 to 2,000 Hz in 4 subjects, and from 125 to 4,000 Hz in 1 subject. The patients claimed to have perceived higher intensities, but with a variable dynamic range from frequency to frequency. The intensity range amounted to between 15 and 40 dB. The obtained data of vibration perception ability are discussed.

摘要

本文探讨了5名极重度聋患者的声音感知能力,这些患者之前在外半规管上形成了“开窗”。先前已有关于前庭装置功能正常的极重度聋患者开窗术后声音和振动感知能力的描述。Wit和Bleeker在鸽子身上进行了该手术的实验。受试者年龄在22至34岁之间。耳聋原因各不相同。他们的听力阈值双侧高于95 dB HL,当用冷热试验测试时,双侧前庭装置兴奋性正常。在所有5名患者中,气导听力阈值保持不变,但振动感知能力显著提高,术后范围在30至45 dB HL之间。4名受试者的频率动态范围为125至2000 Hz,1名受试者为125至4000 Hz。患者声称感知到了更高的强度,但频率间的动态范围各不相同。强度范围在15至40 dB之间。文中讨论了所获得的振动感知能力数据。

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引用本文的文献

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Otol Neurotol. 2017 Aug;38(7):1043-1051. doi: 10.1097/MAO.0000000000001456.
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Conductive hearing loss caused by third-window lesions of the inner ear.内耳第三窗病变导致的传导性听力损失。
Otol Neurotol. 2008 Apr;29(3):282-9. doi: 10.1097/mao.0b013e318161ab24.