Sato Kimitoshi, Yamada Masaru, Shimzu Satoru, Utsuki Satoshi, Konno Shingo, Fujii Kiyotaka, Kan Shinichi
Department of Neurosurgery, Kitasato University School of Medicine, Sagamihara, Kanagawa, Japan.
No Shinkei Geka. 2005 Aug;33(8):805-8.
We report a case of calcified chronic subdural hematoma with unusual magnetic resonance imaging (MRI) findings. A 50-year-old male with no marked medical history presented with fever for two weeks. Computed tomography (CT) revealed a thick, calcified subdural hematoma of low-iso mixed density. MRI showed characteristic signals of hematoma in the acute stage. Total removal of the hematoma by craniotomy was performed after rapid deterioration of consciousness. At surgery, a muddy hematoma and a small amount of pus were seen within the calcified capsule. Diagnosis of infected and calcified chronic subdural hematoma was established. His symptoms were resolved completely within the following few months. As the mechanisms of delayed signal sequence of hematoma on MRI, we speculate existence of tissue hypoxia associated with infection and paramagnetic effects of free radicals and ions associated with inflammation, necrosis and calcification.
我们报告一例具有不寻常磁共振成像(MRI)表现的钙化性慢性硬膜下血肿病例。一名无明显病史的50岁男性出现发热两周。计算机断层扫描(CT)显示为厚的、钙化的低等密度混合性硬膜下血肿。MRI显示血肿在急性期的特征性信号。在意识迅速恶化后,通过开颅手术将血肿完全清除。手术中,在钙化包膜内可见浑浊的血肿和少量脓液。确诊为感染性钙化性慢性硬膜下血肿。在接下来的几个月内,他的症状完全消失。关于MRI上血肿延迟信号序列的机制,我们推测存在与感染相关的组织缺氧以及与炎症、坏死和钙化相关的自由基和离子的顺磁效应。